Comparison of SHANK3 deficiency in animal models : phenotypes, treatment strategies, and translational implications
- Sprache
-
Englisch
- Identifier
-
1299877737
Thema
Autismus; Erbkrankheit; Genetik; Synaptische Transmission; Synapse; Therapie
Beteiligte Personen und Organisationen
Delling, Jan Philipp
Böckers, Tobias M.
Böckers, Tobias M.
- DOI
-
10.18725/OPARU-50043
- URN
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
27.09.2023, 09:58 MESZ
Beteiligte
- Delling, Jan Philipp
- Böckers, Tobias M.
Ähnliche Objekte (12)
Deletion of the autism-associated protein SHANK3 abolishes structural synaptic plasticity after brain trauma
The in vitro effect of oxytocin on synaptic proteins
Phelan McDermid syndrome: multiple sclerosis as a rare but treatable cause for regression—a case report
Synaptic FUS localization during motoneuron development and its accumulation in human ALS synapses
Activation of the medial preoptic area (MPOA) ameliorates loss of maternal behavior in a Shank2 mouse model for autism
Investigation of synaptic ultrastructure in ProSAP/Shank mutant mice
Funktionelle und krankheitsbezogene Charakterisierung des neuronalen Fezzin-Proteins ProSAPiP1
Characterization of the ProSAP1/Shank2 knock-out mouse
Expression des Cylindromatosis Proteins CYLD im zentralen Nervensystem in Maus und Ratte
Charakterisierung der Regulation des Glutamat/Cystin-Antiporters System xc- durch inflammatorische Signalwege in Astrozyten
Characterization of a novel GFP-SHANK3 transgenic mouse model of autism spectrum disorders
Morphologische Analyse dendritischer Spines und synaptischer Kontakte in ProSAP/Shank-Mausmutanten im dorsolateralen Striatum
Deletion of the autism-associated protein SHANK3 abolishes structural synaptic plasticity after brain trauma
The in vitro effect of oxytocin on synaptic proteins
Phelan McDermid syndrome: multiple sclerosis as a rare but treatable cause for regression—a case report
Synaptic FUS localization during motoneuron development and its accumulation in human ALS synapses
Activation of the medial preoptic area (MPOA) ameliorates loss of maternal behavior in a Shank2 mouse model for autism
Investigation of synaptic ultrastructure in ProSAP/Shank mutant mice
Funktionelle und krankheitsbezogene Charakterisierung des neuronalen Fezzin-Proteins ProSAPiP1
Characterization of the ProSAP1/Shank2 knock-out mouse
Expression des Cylindromatosis Proteins CYLD im zentralen Nervensystem in Maus und Ratte
Charakterisierung der Regulation des Glutamat/Cystin-Antiporters System xc- durch inflammatorische Signalwege in Astrozyten
Characterization of a novel GFP-SHANK3 transgenic mouse model of autism spectrum disorders
Morphologische Analyse dendritischer Spines und synaptischer Kontakte in ProSAP/Shank-Mausmutanten im dorsolateralen Striatum
Deletion of the autism-associated protein SHANK3 abolishes structural synaptic plasticity after brain trauma
The in vitro effect of oxytocin on synaptic proteins
Phelan McDermid syndrome: multiple sclerosis as a rare but treatable cause for regression—a case report
Synaptic FUS localization during motoneuron development and its accumulation in human ALS synapses
Activation of the medial preoptic area (MPOA) ameliorates loss of maternal behavior in a Shank2 mouse model for autism
Investigation of synaptic ultrastructure in ProSAP/Shank mutant mice
Funktionelle und krankheitsbezogene Charakterisierung des neuronalen Fezzin-Proteins ProSAPiP1
Characterization of the ProSAP1/Shank2 knock-out mouse
Expression des Cylindromatosis Proteins CYLD im zentralen Nervensystem in Maus und Ratte
Charakterisierung der Regulation des Glutamat/Cystin-Antiporters System xc- durch inflammatorische Signalwege in Astrozyten
Characterization of a novel GFP-SHANK3 transgenic mouse model of autism spectrum disorders