Comparison of SHANK3 deficiency in animal models : phenotypes, treatment strategies, and translational implications

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: In: Journal of Neurodevelopmental Disorders ; 13 (2021). - Art.-Nr. 55. - ISSN 1866-1947. - eISSN 1866-1955

Keyword: Autismus
Erbkrankheit
Genetik
Synaptische Transmission
Synapse
Therapie

Event: Veröffentlichung

(where): Ulm

(who): Universität Ulm

(when): 2021

Creator: Delling, Jan Philipp
Böckers, Tobias M.

DOI: 10.18725/OPARU-50043

URN: urn:nbn:de:bsz:289-oparu-50119-1

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:56 AM CEST

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Associated

Delling, Jan Philipp
Böckers, Tobias M.
Universität Ulm

Time of origin

2021

Other Objects (12)

Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications

Microglia unravel immune-associated alterations in Shank3 deficiency

ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma

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SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids

SHANK3 deficiency leads to myelin defects in the central and peripheral nervous system

Shank3 deficiency elicits autistic-like behaviors by activating p38α in hypothalamic AgRP neurons

Pharmacological modulation of developmental and synaptic phenotypes in human SHANK3 deficient stem cell-derived neuronal models

Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas

Prospective investigation of autism and genotype-phenotype correlations in 22q13 deletion syndrome and SHANK3 deficiency

Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice

Disrupted extracellular matrix and cell cycle genes in autism-associated Shank3 deficiency are targeted by lithium

Shank3 deficiency alters midbrain GABAergic neuron morphology, GABAergic markers and synaptic activity in primary striatal neurons

Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications

Microglia unravel immune-associated alterations in Shank3 deficiency

ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma

Hochschulschrift

SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids

SHANK3 deficiency leads to myelin defects in the central and peripheral nervous system

Shank3 deficiency elicits autistic-like behaviors by activating p38α in hypothalamic AgRP neurons

Pharmacological modulation of developmental and synaptic phenotypes in human SHANK3 deficient stem cell-derived neuronal models

Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas

Prospective investigation of autism and genotype-phenotype correlations in 22q13 deletion syndrome and SHANK3 deficiency

Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice

Disrupted extracellular matrix and cell cycle genes in autism-associated Shank3 deficiency are targeted by lithium

Shank3 deficiency alters midbrain GABAergic neuron morphology, GABAergic markers and synaptic activity in primary striatal neurons

Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications

Microglia unravel immune-associated alterations in Shank3 deficiency

ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma

Hochschulschrift

SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids

SHANK3 deficiency leads to myelin defects in the central and peripheral nervous system

Shank3 deficiency elicits autistic-like behaviors by activating p38α in hypothalamic AgRP neurons

Pharmacological modulation of developmental and synaptic phenotypes in human SHANK3 deficient stem cell-derived neuronal models

Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas

Prospective investigation of autism and genotype-phenotype correlations in 22q13 deletion syndrome and SHANK3 deficiency

Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice

Disrupted extracellular matrix and cell cycle genes in autism-associated Shank3 deficiency are targeted by lithium

Shank3 deficiency alters midbrain GABAergic neuron morphology, GABAergic markers and synaptic activity in primary striatal neurons

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Comparison of SHANK3 deficiency in animal models : phenotypes, treatment strategies, and translational implications

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Other Objects (12)

Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications

Microglia unravel immune-associated alterations in Shank3 deficiency

ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma

SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids

SHANK3 deficiency leads to myelin defects in the central and peripheral nervous system

Shank3 deficiency elicits autistic-like behaviors by activating p38α in hypothalamic AgRP neurons

Pharmacological modulation of developmental and synaptic phenotypes in human SHANK3 deficient stem cell-derived neuronal models

Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas

Prospective investigation of autism and genotype-phenotype correlations in 22q13 deletion syndrome and SHANK3 deficiency

Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice

Disrupted extracellular matrix and cell cycle genes in autism-associated Shank3 deficiency are targeted by lithium

Shank3 deficiency alters midbrain GABAergic neuron morphology, GABAergic markers and synaptic activity in primary striatal neurons

Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications

Microglia unravel immune-associated alterations in Shank3 deficiency

ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma

SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids

SHANK3 deficiency leads to myelin defects in the central and peripheral nervous system

Shank3 deficiency elicits autistic-like behaviors by activating p38α in hypothalamic AgRP neurons

Pharmacological modulation of developmental and synaptic phenotypes in human SHANK3 deficient stem cell-derived neuronal models

Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas

Prospective investigation of autism and genotype-phenotype correlations in 22q13 deletion syndrome and SHANK3 deficiency

Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice

Disrupted extracellular matrix and cell cycle genes in autism-associated Shank3 deficiency are targeted by lithium

Shank3 deficiency alters midbrain GABAergic neuron morphology, GABAergic markers and synaptic activity in primary striatal neurons

Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications

Microglia unravel immune-associated alterations in Shank3 deficiency

ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma

SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids

SHANK3 deficiency leads to myelin defects in the central and peripheral nervous system

Shank3 deficiency elicits autistic-like behaviors by activating p38α in hypothalamic AgRP neurons

Pharmacological modulation of developmental and synaptic phenotypes in human SHANK3 deficient stem cell-derived neuronal models

Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas

Prospective investigation of autism and genotype-phenotype correlations in 22q13 deletion syndrome and SHANK3 deficiency

Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice

Disrupted extracellular matrix and cell cycle genes in autism-associated Shank3 deficiency are targeted by lithium

Shank3 deficiency alters midbrain GABAergic neuron morphology, GABAergic markers and synaptic activity in primary striatal neurons

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