CD4 T cell activation by B cells in human Leishmania (Viannia)infection

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1471-2334

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: CD4 T cell activation by B cells in human Leishmania (Viannia)infection ; volume:14 ; number:1 ; day:25 ; month:2 ; year:2014 ; pages:1-13 ; date:12.2014
BMC infectious diseases ; 14, Heft 1 (25.2.2014), 1-13, 12.2014

Creator: Rodriguez-Pinto, Daniel

Contributor: Saravia, Nancy Gore
McMahon-Pratt, Diane
SpringerLink (Online service)

DOI: 10.1186/1471-2334-14-108

URN: urn:nbn:de:1111-2016030315035

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:23 AM CEST

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Rodriguez-Pinto, Daniel
Saravia, Nancy Gore
McMahon-Pratt, Diane
SpringerLink (Online service)

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CD4 T cell activation by B cells in human Leishmania (Viannia)infection

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Other Objects (12)

Impact of antigen specificity on CD4+T cell activation in chronic HIV-1 infection

Interactions between dendritic cells and CD4+ T cells during Plasmodium infection

LCMV‐specific CD4 T cell dependent polyclonal B‐cell activation upon persistent viral infection is short lived and extrafollicular

PD-1 checkpoint inhibition in Leishmania infection of primary human cells

Intestinal endothelial cells increase HIV infection and latency in resting and activated CD4 + T cells, particularly affecting CCR6 + CD4 + T cells

Metallothionein regulates intracellular zinc signaling during CD4+ T cell activation

HIV‐1 infection of CD4 T cells impairs antigen‐specific B cell function

Oxidative stress, cell activation and viral infection

The role of CD4+T cells in infection-associated immunopathology

Involvement of claudin-7 in HIV infection of CD4(-) cells

Lymphatic endothelial cells promote productive and latent HIV infection in resting CD4+ T cells

Reduced CD4 T cell activation and in vitro susceptibility to HIV-1 infection in exposed uninfected Central Africans

Impact of antigen specificity on CD4+T cell activation in chronic HIV-1 infection

Interactions between dendritic cells and CD4+ T cells during Plasmodium infection

LCMV‐specific CD4 T cell dependent polyclonal B‐cell activation upon persistent viral infection is short lived and extrafollicular

PD-1 checkpoint inhibition in Leishmania infection of primary human cells

Intestinal endothelial cells increase HIV infection and latency in resting and activated CD4 + T cells, particularly affecting CCR6 + CD4 + T cells

Metallothionein regulates intracellular zinc signaling during CD4+ T cell activation

HIV‐1 infection of CD4 T cells impairs antigen‐specific B cell function

Oxidative stress, cell activation and viral infection

The role of CD4+T cells in infection-associated immunopathology

Involvement of claudin-7 in HIV infection of CD4(-) cells

Lymphatic endothelial cells promote productive and latent HIV infection in resting CD4+ T cells

Reduced CD4 T cell activation and in vitro susceptibility to HIV-1 infection in exposed uninfected Central Africans

Impact of antigen specificity on CD4+T cell activation in chronic HIV-1 infection

Interactions between dendritic cells and CD4+ T cells during Plasmodium infection

LCMV‐specific CD4 T cell dependent polyclonal B‐cell activation upon persistent viral infection is short lived and extrafollicular

PD-1 checkpoint inhibition in Leishmania infection of primary human cells

Intestinal endothelial cells increase HIV infection and latency in resting and activated CD4 + T cells, particularly affecting CCR6 + CD4 + T cells

Metallothionein regulates intracellular zinc signaling during CD4+ T cell activation

HIV‐1 infection of CD4 T cells impairs antigen‐specific B cell function

Oxidative stress, cell activation and viral infection

The role of CD4+T cells in infection-associated immunopathology

Involvement of claudin-7 in HIV infection of CD4(-) cells

Lymphatic endothelial cells promote productive and latent HIV infection in resting CD4+ T cells

Reduced CD4 T cell activation and in vitro susceptibility to HIV-1 infection in exposed uninfected Central Africans

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