Altered early immune response after fracture and traumatic brain injury

Language: Englisch

Identifier: 1296552608

Subject

Schädel-Hirn-Trauma; Frakturheilung; Mastzelle

Contributor

Haffner-Luntzer, Melanie
Weber, Birte
Morioka, Kazuhito
Lackner, Ina
Fischer, Verena
Bahney, Chelsea
Ignatius, Anita
Kalbitz, Miriam
Marcucio, Ralph
Miclau, Theodore

DOI: 10.18725/OPARU-49292

URN: http://nbn-resolving.org/urn:nbn:de:bsz:289-oparu-49368-3

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 24.08.2023, 9:54 AM CEST

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Deutsche Nationalbibliothek

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Associated

Haffner-Luntzer, Melanie
Weber, Birte
Morioka, Kazuhito
Lackner, Ina
Fischer, Verena
Bahney, Chelsea
Ignatius, Anita
Kalbitz, Miriam
Marcucio, Ralph
Miclau, Theodore

Other Objects (12)

$A novel mouse model to study fracture healing of the proximal femur$

A novel mouse model to study fracture healing of the proximal femur

Trauma, a matter of the heart : molecular mechanism of post-traumatic cardiac dysfunction

Increased presence of complement factors and mast cells in alveolar bone and tooth resorption

$Differences in fracture healing between female and male C57BL/6J mice$

Differences in fracture healing between female and male C57BL/6J mice

Mast cells drive systemic inflammation and compromised bone repair after trauma

$Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing$

Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing

$Animal models for studying metaphyseal bone fracture healing$

Animal models for studying metaphyseal bone fracture healing

Midkine is elevated after multiple trauma and acts directly on human cardiomyocytes by altering their functionality and metabolism

The role of mast cells in bone metabolism and bone disorders

Complement activation and organ damage after trauma—differential immune response based on surgical treatment strategy

Influence of low-magnitude high-frequency vibration on bone cells and bone regeneration

Myeloid cell-derived catecholamines influence bone turnover and regeneration in mice

$A novel mouse model to study fracture healing of the proximal femur$

A novel mouse model to study fracture healing of the proximal femur

Trauma, a matter of the heart : molecular mechanism of post-traumatic cardiac dysfunction

Increased presence of complement factors and mast cells in alveolar bone and tooth resorption

$Differences in fracture healing between female and male C57BL/6J mice$

Differences in fracture healing between female and male C57BL/6J mice

Mast cells drive systemic inflammation and compromised bone repair after trauma

$Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing$

Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing

$Animal models for studying metaphyseal bone fracture healing$

Animal models for studying metaphyseal bone fracture healing

Midkine is elevated after multiple trauma and acts directly on human cardiomyocytes by altering their functionality and metabolism

The role of mast cells in bone metabolism and bone disorders

Complement activation and organ damage after trauma—differential immune response based on surgical treatment strategy

Influence of low-magnitude high-frequency vibration on bone cells and bone regeneration

Myeloid cell-derived catecholamines influence bone turnover and regeneration in mice

$A novel mouse model to study fracture healing of the proximal femur$

A novel mouse model to study fracture healing of the proximal femur

Trauma, a matter of the heart : molecular mechanism of post-traumatic cardiac dysfunction

Increased presence of complement factors and mast cells in alveolar bone and tooth resorption

$Differences in fracture healing between female and male C57BL/6J mice$

Differences in fracture healing between female and male C57BL/6J mice

Mast cells drive systemic inflammation and compromised bone repair after trauma

$Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing$

Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing

$Animal models for studying metaphyseal bone fracture healing$

Animal models for studying metaphyseal bone fracture healing

Midkine is elevated after multiple trauma and acts directly on human cardiomyocytes by altering their functionality and metabolism

The role of mast cells in bone metabolism and bone disorders

Complement activation and organ damage after trauma—differential immune response based on surgical treatment strategy

Influence of low-magnitude high-frequency vibration on bone cells and bone regeneration

Myeloid cell-derived catecholamines influence bone turnover and regeneration in mice

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Altered early immune response after fracture and traumatic brain injury

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Associated

Other Objects (12)

A novel mouse model to study fracture healing of the proximal femur

Trauma, a matter of the heart : molecular mechanism of post-traumatic cardiac dysfunction

Increased presence of complement factors and mast cells in alveolar bone and tooth resorption

Differences in fracture healing between female and male C57BL/6J mice

Mast cells drive systemic inflammation and compromised bone repair after trauma

Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing

Animal models for studying metaphyseal bone fracture healing

Midkine is elevated after multiple trauma and acts directly on human cardiomyocytes by altering their functionality and metabolism

The role of mast cells in bone metabolism and bone disorders

Complement activation and organ damage after trauma—differential immune response based on surgical treatment strategy

Influence of low-magnitude high-frequency vibration on bone cells and bone regeneration

Myeloid cell-derived catecholamines influence bone turnover and regeneration in mice

A novel mouse model to study fracture healing of the proximal femur

Trauma, a matter of the heart : molecular mechanism of post-traumatic cardiac dysfunction

Increased presence of complement factors and mast cells in alveolar bone and tooth resorption

Differences in fracture healing between female and male C57BL/6J mice

Mast cells drive systemic inflammation and compromised bone repair after trauma

Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing

Animal models for studying metaphyseal bone fracture healing

Midkine is elevated after multiple trauma and acts directly on human cardiomyocytes by altering their functionality and metabolism

The role of mast cells in bone metabolism and bone disorders

Complement activation and organ damage after trauma—differential immune response based on surgical treatment strategy

Influence of low-magnitude high-frequency vibration on bone cells and bone regeneration

Myeloid cell-derived catecholamines influence bone turnover and regeneration in mice

A novel mouse model to study fracture healing of the proximal femur

Trauma, a matter of the heart : molecular mechanism of post-traumatic cardiac dysfunction

Increased presence of complement factors and mast cells in alveolar bone and tooth resorption

Differences in fracture healing between female and male C57BL/6J mice

Mast cells drive systemic inflammation and compromised bone repair after trauma

Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing

Animal models for studying metaphyseal bone fracture healing

Midkine is elevated after multiple trauma and acts directly on human cardiomyocytes by altering their functionality and metabolism

The role of mast cells in bone metabolism and bone disorders

Complement activation and organ damage after trauma—differential immune response based on surgical treatment strategy

Influence of low-magnitude high-frequency vibration on bone cells and bone regeneration

Myeloid cell-derived catecholamines influence bone turnover and regeneration in mice

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