Hochschulschrift

Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: In: Frontiers in Bioengineering and Biotechnology ; 9 (2021). - Art.-Nr. 782355. - eISSN 2296-4185

Keyword: Osteoblast
Frakturheilung
Schwingung
Therapie
Osteoporose
Menopause
Östrogenrezeptor
Frakturheilung

Event: Veröffentlichung

(where): Ulm

(who): Universität Ulm

(when): 2021

Creator: Steppe, Lena
Krüger, Benjamin Thilo
Tschaffon, Miriam Eva Angelica
Fischer, Verena
Tuckermann, Jan Peter Gottfried
Ignatius, Anita
Haffner-Luntzer, Melanie

DOI: 10.18725/OPARU-42007

URN: urn:nbn:de:bsz:289-oparu-42083-4

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:35 AM CEST

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Hochschulschrift

Associated

Steppe, Lena
Krüger, Benjamin Thilo
Tschaffon, Miriam Eva Angelica
Fischer, Verena
Tuckermann, Jan Peter Gottfried
Ignatius, Anita
Haffner-Luntzer, Melanie
Universität Ulm

Time of origin

2021

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$Estrogen receptor α signaling in osteoblast lineage-specific cells is crucial for bone homeostasis and mechanotransduction in bone fracture healing$

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Estrogen receptor α signaling in osteoblast lineage-specific cells is crucial for bone homeostasis and mechanotransduction in bone fracture healing

P53 and Beta-Catenin Activity during Estrogen treatment of Osteoblasts

Revealing hidden information in osteoblast’s mechanotransduction through analysis of time patterns of critical events

Mechanotransduction pathways in articular chondrocytes and the emerging role of estrogen receptor-α

β1 integrin-dependent mechanotransduction induces angiocrine signaling required to promote liver growth and survival

Human osteoblasts exhibit sexual dimorphism in their response to estrogen on microstructured titanium surfaces

Mechanotransduction is required for establishing and maintaining mature inner hair cells and regulating efferent innervation

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Estrogen Plus Estrogen Receptor Antagonists Alter Mineral Production by Osteoblasts In Vitro

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Characterization of estrogen receptor α [alpha] in mouse osteoblasts

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Characterization of estrogen receptor α [alpha] in mouse osteoblasts

$Estrogen receptor α signaling in osteoblast lineage-specific cells is crucial for bone homeostasis and mechanotransduction in bone fracture healing$

Hochschulschrift

Estrogen receptor α signaling in osteoblast lineage-specific cells is crucial for bone homeostasis and mechanotransduction in bone fracture healing

P53 and Beta-Catenin Activity during Estrogen treatment of Osteoblasts

Revealing hidden information in osteoblast’s mechanotransduction through analysis of time patterns of critical events

Mechanotransduction pathways in articular chondrocytes and the emerging role of estrogen receptor-α

β1 integrin-dependent mechanotransduction induces angiocrine signaling required to promote liver growth and survival

Human osteoblasts exhibit sexual dimorphism in their response to estrogen on microstructured titanium surfaces

Mechanotransduction is required for establishing and maintaining mature inner hair cells and regulating efferent innervation

Aging, Osteocytes, and Mechanotransduction

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Estrogen receptor α signaling in osteoblasts is required for mechanotransduction in bone fracture healing

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Other Objects (12)

Cellular mechanotransduction of human osteoblasts in microgravity

Estrogen Plus Estrogen Receptor Antagonists Alter Mineral Production by Osteoblasts In Vitro

Characterization of estrogen receptor α [alpha] in mouse osteoblasts

Characterization of estrogen receptor α [alpha] in mouse osteoblasts

Estrogen receptor α signaling in osteoblast lineage-specific cells is crucial for bone homeostasis and mechanotransduction in bone fracture healing

P53 and Beta-Catenin Activity during Estrogen treatment of Osteoblasts

Revealing hidden information in osteoblast’s mechanotransduction through analysis of time patterns of critical events

Mechanotransduction pathways in articular chondrocytes and the emerging role of estrogen receptor-α

β1 integrin-dependent mechanotransduction induces angiocrine signaling required to promote liver growth and survival

Human osteoblasts exhibit sexual dimorphism in their response to estrogen on microstructured titanium surfaces

Mechanotransduction is required for establishing and maintaining mature inner hair cells and regulating efferent innervation

Aging, Osteocytes, and Mechanotransduction

Cellular mechanotransduction of human osteoblasts in microgravity

Estrogen Plus Estrogen Receptor Antagonists Alter Mineral Production by Osteoblasts In Vitro

Characterization of estrogen receptor α [alpha] in mouse osteoblasts

Characterization of estrogen receptor α [alpha] in mouse osteoblasts

Estrogen receptor α signaling in osteoblast lineage-specific cells is crucial for bone homeostasis and mechanotransduction in bone fracture healing

P53 and Beta-Catenin Activity during Estrogen treatment of Osteoblasts

Revealing hidden information in osteoblast’s mechanotransduction through analysis of time patterns of critical events

Mechanotransduction pathways in articular chondrocytes and the emerging role of estrogen receptor-α

β1 integrin-dependent mechanotransduction induces angiocrine signaling required to promote liver growth and survival

Human osteoblasts exhibit sexual dimorphism in their response to estrogen on microstructured titanium surfaces

Mechanotransduction is required for establishing and maintaining mature inner hair cells and regulating efferent innervation

Aging, Osteocytes, and Mechanotransduction

Cellular mechanotransduction of human osteoblasts in microgravity

Estrogen Plus Estrogen Receptor Antagonists Alter Mineral Production by Osteoblasts In Vitro

Characterization of estrogen receptor α [alpha] in mouse osteoblasts

Characterization of estrogen receptor α [alpha] in mouse osteoblasts

Estrogen receptor α signaling in osteoblast lineage-specific cells is crucial for bone homeostasis and mechanotransduction in bone fracture healing

P53 and Beta-Catenin Activity during Estrogen treatment of Osteoblasts

Revealing hidden information in osteoblast’s mechanotransduction through analysis of time patterns of critical events

Mechanotransduction pathways in articular chondrocytes and the emerging role of estrogen receptor-α

β1 integrin-dependent mechanotransduction induces angiocrine signaling required to promote liver growth and survival

Human osteoblasts exhibit sexual dimorphism in their response to estrogen on microstructured titanium surfaces

Mechanotransduction is required for establishing and maintaining mature inner hair cells and regulating efferent innervation

Aging, Osteocytes, and Mechanotransduction

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