Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-1723
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas ; volume:13 ; number:1 ; day:28 ; month:11 ; year:2022 ; pages:1-13 ; date:12.2022
Nature Communications ; 13, Heft 1 (28.11.2022), 1-13, 12.2022
- Creator
-
Kern, Joseph G.
Tilston-Lunel, Andrew M.
Federico, Anthony
Ning, Boting
Mueller, Amy
Peppler, Grace B.
Stampouloglou, Eleni
Cheng, Nan
Johnson, Randy L.
Lenburg, Marc E.
Beane, Jennifer E.
Monti, Stefano
Varelas, Xaralabos
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-022-34864-8
- URN
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urn:nbn:de:101:1-2023020509015095227527
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:53 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Kern, Joseph G.
- Tilston-Lunel, Andrew M.
- Federico, Anthony
- Ning, Boting
- Mueller, Amy
- Peppler, Grace B.
- Stampouloglou, Eleni
- Cheng, Nan
- Johnson, Randy L.
- Lenburg, Marc E.
- Beane, Jennifer E.
- Monti, Stefano
- Varelas, Xaralabos
- SpringerLink (Online service)
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Association between mammographic density and basal-like and luminal A breast cancer subtypes
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The functional loss of the retinoblastoma tumour suppressor is a common event in basal-like and luminal B breast carcinomas
Isolation and characterization of a new basal-like luminal progenitor in human breast tissue
Lineage plasticity enables low-ER luminal tumors to evolve and gain basal-like traits
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Ductal keratin 15+ luminal progenitors in normal breast exhibit a basal-like breast cancer transcriptomic signature
Inhibition of SHP2 in basal-like and triple-negative breast cells induces basal-to-luminal transition, hormone dependency, and sensitivity to anti-hormone treatment
Expression of thyroid transcription factor-1 is associated with a basal-like phenotype in breast carcinomas
Oxidative stress and counteracting mechanisms in hormone receptor positive, triple-negative and basal-like breast carcinomas
Race-associated biological differences among luminal A and basal-like breast cancers in the Carolina Breast Cancer Study
Identification of key genes unique to the luminal a and basal-like breast cancer subtypes via bioinformatic analysis
Association between mammographic density and basal-like and luminal A breast cancer subtypes
A comprehensive morphological study for basal-like breast carcinomas with comparison to nonbasal-like carcinomas
The functional loss of the retinoblastoma tumour suppressor is a common event in basal-like and luminal B breast carcinomas
Isolation and characterization of a new basal-like luminal progenitor in human breast tissue
Lineage plasticity enables low-ER luminal tumors to evolve and gain basal-like traits
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Ductal keratin 15+ luminal progenitors in normal breast exhibit a basal-like breast cancer transcriptomic signature
Inhibition of SHP2 in basal-like and triple-negative breast cells induces basal-to-luminal transition, hormone dependency, and sensitivity to anti-hormone treatment
Expression of thyroid transcription factor-1 is associated with a basal-like phenotype in breast carcinomas
Oxidative stress and counteracting mechanisms in hormone receptor positive, triple-negative and basal-like breast carcinomas
Race-associated biological differences among luminal A and basal-like breast cancers in the Carolina Breast Cancer Study