Inhibition of SHP2 in basal-like and triple-negative breast cells induces basal-to-luminal transition, hormone dependency, and sensitivity to anti-hormone treatment
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2407
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Inhibition of SHP2 in basal-like and triple-negative breast cells induces basal-to-luminal transition, hormone dependency, and sensitivity to anti-hormone treatment ; volume:15 ; number:1 ; day:8 ; month:3 ; year:2015 ; pages:1-12 ; date:12.2015
BMC cancer ; 15, Heft 1 (8.3.2015), 1-12, 12.2015
- Creator
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Zhao, Hua
- Contributor
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Agazie, Yehenew M.
SpringerLink (Online service)
- DOI
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10.1186/s12885-015-1131-2
- URN
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urn:nbn:de:1111-2016110616167
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:50 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Zhao, Hua
- Agazie, Yehenew M.
- SpringerLink (Online service)
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Hormones induce the formation of luminal-derived basal cells in the mammary gland
Association between mammographic density and basal-like and luminal A breast cancer subtypes
Isolation and characterization of a new basal-like luminal progenitor in human breast tissue
Lineage plasticity enables low-ER luminal tumors to evolve and gain basal-like traits
Oxidative stress and counteracting mechanisms in hormone receptor positive, triple-negative and basal-like breast carcinomas
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Ductal keratin 15+ luminal progenitors in normal breast exhibit a basal-like breast cancer transcriptomic signature
Race-associated biological differences among luminal A and basal-like breast cancers in the Carolina Breast Cancer Study
Luminal and basal-like breast cancer cells show increased migration induced by hypoxia, mediated by an autocrine mechanism
Identification of key genes unique to the luminal a and basal-like breast cancer subtypes via bioinformatic analysis
SHP2 acts both upstream and downstream of multiple receptor tyrosine kinases to promote basal-like and triple-negative breast cancer
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Hormones induce the formation of luminal-derived basal cells in the mammary gland
Association between mammographic density and basal-like and luminal A breast cancer subtypes
Isolation and characterization of a new basal-like luminal progenitor in human breast tissue
Lineage plasticity enables low-ER luminal tumors to evolve and gain basal-like traits
Oxidative stress and counteracting mechanisms in hormone receptor positive, triple-negative and basal-like breast carcinomas
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Ductal keratin 15+ luminal progenitors in normal breast exhibit a basal-like breast cancer transcriptomic signature
Race-associated biological differences among luminal A and basal-like breast cancers in the Carolina Breast Cancer Study
Luminal and basal-like breast cancer cells show increased migration induced by hypoxia, mediated by an autocrine mechanism
Identification of key genes unique to the luminal a and basal-like breast cancer subtypes via bioinformatic analysis