Expression of thyroid transcription factor-1 is associated with a basal-like phenotype in breast carcinomas
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1746-1596
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Expression of thyroid transcription factor-1 is associated with a basal-like phenotype in breast carcinomas ; volume:8 ; number:1 ; day:15 ; month:5 ; year:2013 ; pages:1-6 ; date:12.2013
Diagnostic pathology ; 8, Heft 1 (15.5.2013), 1-6, 12.2013
- Creator
-
Klingen, Tor A.
- Contributor
-
Chen, Ying
Suhrke, Pål
Stefansson, Ingunn M.
Gundersen, Marian D.
Akslen, Lars A.
SpringerLink (Online service)
- DOI
-
10.1186/1746-1596-8-80
- URN
-
urn:nbn:de:1111-2016080742320
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:48 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Klingen, Tor A.
- Chen, Ying
- Suhrke, Pål
- Stefansson, Ingunn M.
- Gundersen, Marian D.
- Akslen, Lars A.
- SpringerLink (Online service)
Other Objects (12)
Molecular studies on a Dictyostelium homolog of the tafazzin gene, the cause of Barth syndrome in humans
Big data technology for computer intrusion detection
One Hundred Years of Chinese Literature Translation in Bulgaria
Molecular studies on a Dictyostelium homolog of the tafazzin gene, the cause of Barth syndrome in humans
The hot spots and frontiers of research on the Grand Canal Culture Belt in China: Literature and academic trends
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
A comprehensive morphological study for basal-like breast carcinomas with comparison to nonbasal-like carcinomas
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC
WNT5B governs the phenotype of basal-like breast cancer by activating WNT signaling
Learning while setting precedents
Rhetoric in legislative bargaining with asymmetric information
Molecular studies on a Dictyostelium homolog of the tafazzin gene, the cause of Barth syndrome in humans
Big data technology for computer intrusion detection
One Hundred Years of Chinese Literature Translation in Bulgaria
Molecular studies on a Dictyostelium homolog of the tafazzin gene, the cause of Barth syndrome in humans
The hot spots and frontiers of research on the Grand Canal Culture Belt in China: Literature and academic trends
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
A comprehensive morphological study for basal-like breast carcinomas with comparison to nonbasal-like carcinomas
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC
WNT5B governs the phenotype of basal-like breast cancer by activating WNT signaling
Learning while setting precedents
Rhetoric in legislative bargaining with asymmetric information
Molecular studies on a Dictyostelium homolog of the tafazzin gene, the cause of Barth syndrome in humans
Big data technology for computer intrusion detection
One Hundred Years of Chinese Literature Translation in Bulgaria
Molecular studies on a Dictyostelium homolog of the tafazzin gene, the cause of Barth syndrome in humans
The hot spots and frontiers of research on the Grand Canal Culture Belt in China: Literature and academic trends
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
A comprehensive morphological study for basal-like breast carcinomas with comparison to nonbasal-like carcinomas
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC
WNT5B governs the phenotype of basal-like breast cancer by activating WNT signaling
Learning while setting precedents