Hochschulschrift
CD64-mediated specific elimination of M1-polarized macrophages : implications for therapeutic intervention in chronic inflammatory diseases
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
Aachen, Techn. Hochsch., Diss., 2013
- Schlagwort
-
Makrophage , Entzündung , Immuntherapie
Immunologie
Immunsystem
Lymphozyt
Leukozyt
Immunität
Resistenz
Immunchemie
- Ereignis
-
Veröffentlichung
- (wo)
-
Aachen
- (wer)
-
Hochschulbibliothek der Rheinisch-Westfälischen Technischen Hochschule Aachen
- (wann)
-
2013
- Urheber
- URN
-
urn:nbn:de:hbz:82-opus-47054
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.03.2025, 13:53 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Objekttyp
- Hochschulschrift
Beteiligte
- Hristodorov, Dmitrij
- Hochschulbibliothek der Rheinisch-Westfälischen Technischen Hochschule Aachen
Entstanden
- 2013
Ähnliche Objekte (12)
CD64-mediated specific elimination of M1-polarized macrophages : implications for therapeutic intervention in chronic inflammatory diseases
PACAP regulates VPAC1 expression, inflammatory processes and lipid homeostasis in M1- and M2-macrophages
The Impact of Rubella Virus Infection on a Secondary Inflammatory Response in Polarized Human Macrophages
The Transcription Factor STAT6 Mediates Direct Repression of Inflammatory Enhancers and Limits Activation of Alternatively Polarized Macrophages
The influence of danger and resolution signals on innate immune memory in human primary monocytes and in M1 or M2 polarized macrophages
Bidirectional Interaction of Differently Activated and Polarized Monocytes/Macrophages and Stromal Cells in Context of Tissue Remodeling in Chronic Inflammatory Disease
The Contribution of Macrophages to Inflammatory Corneal Lymphangiogenesis
GR actions in stromal cells suppress inflammatory arthritis by induction of anti-inflammatory macrophages
IKKα promotes intestinal tumorigenesis by limiting recruitment of M1-like polarized myeloid cells
Glucocorticoid activation of anti-inflammatory macrophages protects against insulin resistance
Hypoxia potentiates palmitate-induced pro-inflammatory activation of primary human macrophages
CXCL10 Controls Inflammatory Pain via Opioid Peptide- Containing Macrophages in Electroacupuncture
CD64-mediated specific elimination of M1-polarized macrophages : implications for therapeutic intervention in chronic inflammatory diseases
PACAP regulates VPAC1 expression, inflammatory processes and lipid homeostasis in M1- and M2-macrophages
The Impact of Rubella Virus Infection on a Secondary Inflammatory Response in Polarized Human Macrophages
The Transcription Factor STAT6 Mediates Direct Repression of Inflammatory Enhancers and Limits Activation of Alternatively Polarized Macrophages
The influence of danger and resolution signals on innate immune memory in human primary monocytes and in M1 or M2 polarized macrophages
Bidirectional Interaction of Differently Activated and Polarized Monocytes/Macrophages and Stromal Cells in Context of Tissue Remodeling in Chronic Inflammatory Disease
The Contribution of Macrophages to Inflammatory Corneal Lymphangiogenesis
GR actions in stromal cells suppress inflammatory arthritis by induction of anti-inflammatory macrophages
IKKα promotes intestinal tumorigenesis by limiting recruitment of M1-like polarized myeloid cells
Glucocorticoid activation of anti-inflammatory macrophages protects against insulin resistance
Hypoxia potentiates palmitate-induced pro-inflammatory activation of primary human macrophages
CXCL10 Controls Inflammatory Pain via Opioid Peptide- Containing Macrophages in Electroacupuncture
CD64-mediated specific elimination of M1-polarized macrophages : implications for therapeutic intervention in chronic inflammatory diseases
PACAP regulates VPAC1 expression, inflammatory processes and lipid homeostasis in M1- and M2-macrophages
The Impact of Rubella Virus Infection on a Secondary Inflammatory Response in Polarized Human Macrophages
The Transcription Factor STAT6 Mediates Direct Repression of Inflammatory Enhancers and Limits Activation of Alternatively Polarized Macrophages
The influence of danger and resolution signals on innate immune memory in human primary monocytes and in M1 or M2 polarized macrophages
Bidirectional Interaction of Differently Activated and Polarized Monocytes/Macrophages and Stromal Cells in Context of Tissue Remodeling in Chronic Inflammatory Disease
The Contribution of Macrophages to Inflammatory Corneal Lymphangiogenesis
GR actions in stromal cells suppress inflammatory arthritis by induction of anti-inflammatory macrophages
IKKα promotes intestinal tumorigenesis by limiting recruitment of M1-like polarized myeloid cells
Glucocorticoid activation of anti-inflammatory macrophages protects against insulin resistance
Hypoxia potentiates palmitate-induced pro-inflammatory activation of primary human macrophages