Hypoxia potentiates palmitate-induced pro-inflammatory activation of primary human macrophages
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Erschienen in
-
In: Journal of biological chemistry, 291.2016, 1, S. 413-424
- Ereignis
-
Veröffentlichung
- (wo)
-
Frankfurt am Main
- (wer)
-
Universitätsbibliothek Johann Christian Senckenberg
- (wann)
-
2021
- Urheber
-
Snodgrass, Ryan G.
Boß, Marcel
Zezina, Ekaterina
Weigert, Andreas
Dehne, Nathalie
Fleming, Ingrid
Brüne, Bernhard
Namgaladze, Dmitry
- DOI
-
10.1074/jbc.M115.686709
- URN
-
urn:nbn:de:hebis:30:3-772348
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.03.2025, 13:47 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Snodgrass, Ryan G.
- Boß, Marcel
- Zezina, Ekaterina
- Weigert, Andreas
- Dehne, Nathalie
- Fleming, Ingrid
- Brüne, Bernhard
- Namgaladze, Dmitry
- Universitätsbibliothek Johann Christian Senckenberg
Entstanden
- 2021
Ähnliche Objekte (12)
Mitofusin 2 deficiency causes pro-inflammatory effects in human primary macrophages
Regulation der Thrombozytenaggregation durch cGMP
miR-193a-3p increases glycolysis under hypoxia by facilitating Akt phosphorylation and PFKFB3 activation in human macrophages
Pharmacological activation of p53 during human monocyte to macrophage differentiation attenuates their pro-inflammatory activation by TLR4, TLR7 and TLR8 agonists
Mitochondrial composition and function under the control of hypoxia
Regulation and functions of 15-lipoxygenases in human macrophages
A graphical journey through iron metabolism, microRNAs, and hypoxia in ferroptosis
AICAR inhibits NFκB DNA binding independently of AMPK to attenuate LPS-triggered inflammatory responses in human macrophages
The role of HuR in the crosstalk between tumor cells and macrophages
The PGE2/cAMP signaling axis in macrophages during inflammation
MicroRNA - a tumor trojan horse for tumor-associated macrophages
Hyperoxia/Hypoxia Exposure Primes a Sustained Pro-Inflammatory Profile of Preterm Infant Macrophages Upon LPS Stimulation
Mitofusin 2 deficiency causes pro-inflammatory effects in human primary macrophages
Regulation der Thrombozytenaggregation durch cGMP
miR-193a-3p increases glycolysis under hypoxia by facilitating Akt phosphorylation and PFKFB3 activation in human macrophages
Pharmacological activation of p53 during human monocyte to macrophage differentiation attenuates their pro-inflammatory activation by TLR4, TLR7 and TLR8 agonists
Mitochondrial composition and function under the control of hypoxia
Regulation and functions of 15-lipoxygenases in human macrophages
A graphical journey through iron metabolism, microRNAs, and hypoxia in ferroptosis
AICAR inhibits NFκB DNA binding independently of AMPK to attenuate LPS-triggered inflammatory responses in human macrophages
The role of HuR in the crosstalk between tumor cells and macrophages
The PGE2/cAMP signaling axis in macrophages during inflammation
MicroRNA - a tumor trojan horse for tumor-associated macrophages
Hyperoxia/Hypoxia Exposure Primes a Sustained Pro-Inflammatory Profile of Preterm Infant Macrophages Upon LPS Stimulation
Mitofusin 2 deficiency causes pro-inflammatory effects in human primary macrophages
Regulation der Thrombozytenaggregation durch cGMP
miR-193a-3p increases glycolysis under hypoxia by facilitating Akt phosphorylation and PFKFB3 activation in human macrophages
Pharmacological activation of p53 during human monocyte to macrophage differentiation attenuates their pro-inflammatory activation by TLR4, TLR7 and TLR8 agonists
Mitochondrial composition and function under the control of hypoxia
Regulation and functions of 15-lipoxygenases in human macrophages
A graphical journey through iron metabolism, microRNAs, and hypoxia in ferroptosis
AICAR inhibits NFκB DNA binding independently of AMPK to attenuate LPS-triggered inflammatory responses in human macrophages
The role of HuR in the crosstalk between tumor cells and macrophages
The PGE2/cAMP signaling axis in macrophages during inflammation
MicroRNA - a tumor trojan horse for tumor-associated macrophages