Histidine containing dipeptides protect epithelial and endothelial cell barriers from methylglyoxal induced injury
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Histidine containing dipeptides protect epithelial and endothelial cell barriers from methylglyoxal induced injury ; volume:14 ; number:1 ; day:4 ; month:11 ; year:2024 ; pages:1-12 ; date:12.2024
Scientific reports ; 14, Heft 1 (4.11.2024), 1-12, 12.2024
- Creator
-
Wetzel, Charlotte
Gallenstein, Nadia
Peters, Verena
Fleming, Thomas
Marinovic, Iva
Bodenschatz, Alea
Du, Zhiwei
Küper, Katharina
Dallanoce, Clelia
Aldini, Giancarlo
Schmoch, Thomas
Brenner, Thorsten
Weigand, Markus Alexander
Zarogiannis, Sotirios G.
Schmitt, Claus Peter
Bartosova, Maria
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41598-024-77891-9
- URN
-
urn:nbn:de:101:1-2501222120264.547756438943
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:35 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wetzel, Charlotte
- Gallenstein, Nadia
- Peters, Verena
- Fleming, Thomas
- Marinovic, Iva
- Bodenschatz, Alea
- Du, Zhiwei
- Küper, Katharina
- Dallanoce, Clelia
- Aldini, Giancarlo
- Schmoch, Thomas
- Brenner, Thorsten
- Weigand, Markus Alexander
- Zarogiannis, Sotirios G.
- Schmitt, Claus Peter
- Bartosova, Maria
- SpringerLink (Online service)
Other Objects (12)
Methylglyoxal induced endothelial dysfunction via loss of proliferation
Pathophysiology of methylglyoxal-associated endothelial damage in experimental sepsis
Phenylpyrrole fungicides act on triosephosphate isomerase to induce methylglyoxal stress and alter hybrid histidine kinase activity
Endothelial cell and macrophage responses induced by methylglyoxal : potential role of mTORC1
Endothelial Na+/H+ exchanger NHE1 participates in redox-sensitive leukocyte recruitment triggered by methylglyoxal
High-volume hemofiltration does not protect human kidney endothelial and tubular epithelial cells from septic plasma-induced injury
Statins protect diabetic myocardial microvascular endothelial cells from injury
Activation of the unfolded protein response in high glucose treated endothelial cells is mediated by methylglyoxal
Dietary antioxidants protect gut epithelial cells from oxidant-induced apoptosis
Inhibition of HIF prolyl 4-hydroxylases protect endothelial cells from apoptosis
Inhibition of HIF prolyl 4-hydroxylases protect endothelial cells from apoptosis
Histidine, 3.. Bermuda 1982
Methylglyoxal induced endothelial dysfunction via loss of proliferation
Pathophysiology of methylglyoxal-associated endothelial damage in experimental sepsis
Phenylpyrrole fungicides act on triosephosphate isomerase to induce methylglyoxal stress and alter hybrid histidine kinase activity
Endothelial cell and macrophage responses induced by methylglyoxal : potential role of mTORC1
Endothelial Na+/H+ exchanger NHE1 participates in redox-sensitive leukocyte recruitment triggered by methylglyoxal
High-volume hemofiltration does not protect human kidney endothelial and tubular epithelial cells from septic plasma-induced injury
Statins protect diabetic myocardial microvascular endothelial cells from injury
Activation of the unfolded protein response in high glucose treated endothelial cells is mediated by methylglyoxal
Dietary antioxidants protect gut epithelial cells from oxidant-induced apoptosis
Inhibition of HIF prolyl 4-hydroxylases protect endothelial cells from apoptosis
Inhibition of HIF prolyl 4-hydroxylases protect endothelial cells from apoptosis
Histidine, 3.. Bermuda 1982
Methylglyoxal induced endothelial dysfunction via loss of proliferation
Pathophysiology of methylglyoxal-associated endothelial damage in experimental sepsis
Phenylpyrrole fungicides act on triosephosphate isomerase to induce methylglyoxal stress and alter hybrid histidine kinase activity
Endothelial cell and macrophage responses induced by methylglyoxal : potential role of mTORC1
Endothelial Na+/H+ exchanger NHE1 participates in redox-sensitive leukocyte recruitment triggered by methylglyoxal
High-volume hemofiltration does not protect human kidney endothelial and tubular epithelial cells from septic plasma-induced injury
Statins protect diabetic myocardial microvascular endothelial cells from injury
Activation of the unfolded protein response in high glucose treated endothelial cells is mediated by methylglyoxal
Dietary antioxidants protect gut epithelial cells from oxidant-induced apoptosis
Inhibition of HIF prolyl 4-hydroxylases protect endothelial cells from apoptosis
Inhibition of HIF prolyl 4-hydroxylases protect endothelial cells from apoptosis