Changes in glutamic oxaloacetic transaminase 2 during rat physiological and pathological cardiomyocyte hypertrophy
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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1 Online-Ressource.
- Sprache
-
Englisch
- Erschienen in
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Changes in glutamic oxaloacetic transaminase 2 during rat physiological and pathological cardiomyocyte hypertrophy ; volume:23 ; number:1 ; day:5 ; month:12 ; year:2023 ; pages:1-8 ; date:12.2023
BMC cardiovascular disorders ; 23, Heft 1 (5.12.2023), 1-8, 12.2023
- Klassifikation
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Medizin, Gesundheit
- Urheber
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Liu, Xin
Li, Xiaolu
Zhou, Haotan
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
-
10.1186/s12872-023-03648-3
- URN
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urn:nbn:de:101:1-2024022113491277463431
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 11:00 MESZ
Datenpartner
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Beteiligte
- Liu, Xin
- Li, Xiaolu
- Zhou, Haotan
- SpringerLink (Online service)
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LOC102549726/miR-760-3p network is involved in the progression of ISO-induced pathological cardiomyocyte hypertrophy via endoplasmic reticulum stress
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The malate aspartate shuttle in T cell metabolic fitness: Glutamic oxaloacetic transaminase 1 antagonizes T cell exhaustion
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Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival
The functional role of m6A demethylase ALKBH5 in cardiomyocyte hypertrophy
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Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
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