Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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Online-Ressource
- Sprache
-
Englisch
- Erschienen in
-
In: Scientific Reports 6:35758 (2016). DOI: 10.1038/srep35758
- Ereignis
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Veröffentlichung
- (wo)
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Würzburg
- (wer)
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Universität Würzburg
- (wann)
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2019
- URN
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urn:nbn:de:bvb:20-opus-167525
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.03.2025, 13:51 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
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Beteiligte
- Bernt, Alexander
- Rangrez, Ashraf Y.
- Eden, Matthias
- Jungmann, Andreas
- Katz, Sylvia
- Rohr, Claudia
- Müller, Oliver J.
- Katus, Hugo
- Sossalla, Samuel Tobias
- Williams, Tatjana
- Ritter, Oliver
- Frank, Derk
- Frey, Norbert
- Universität Würzburg
Entstanden
- 2019
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Three-dimensional human pluripotent stem cell-derived cardiomyocyte models for investigating cardiac hypertrophy
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Amlexanox and forskolin prevents isoproterenol-induced cardiomyopathy by subduing cardiomyocyte hypertrophy and maladaptive inflammatory responses
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Calcineurin-independent NFATc1 signaling is essential for survival of Burkitt lymphoma cells
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The acute phase protein alpha-2-macroglobulin induces rat ventricular cardiomyocyte hypertrophy via ERK1, 2 and PI3 kinase, akt pathways
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Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
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Three-dimensional human pluripotent stem cell-derived cardiomyocyte models for investigating cardiac hypertrophy
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Altered cellular protein quality control system modulates cardiomyocyte function in volume overload-induced hypertrophy
Amlexanox and forskolin prevents isoproterenol-induced cardiomyopathy by subduing cardiomyocyte hypertrophy and maladaptive inflammatory responses
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Using iPSC-CMs to develop a new, 2D in vitro culture-based model for cardiomyocyte hypertrophy
Calcineurin-independent NFATc1 signaling is essential for survival of Burkitt lymphoma cells
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