Changes in glutamic oxaloacetic transaminase 2 during rat physiological and pathological cardiomyocyte hypertrophy
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Changes in glutamic oxaloacetic transaminase 2 during rat physiological and pathological cardiomyocyte hypertrophy ; volume:23 ; number:1 ; day:5 ; month:12 ; year:2023 ; pages:1-8 ; date:12.2023
BMC cardiovascular disorders ; 23, Heft 1 (5.12.2023), 1-8, 12.2023
- Classification
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Medizin, Gesundheit
- Creator
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Liu, Xin
Li, Xiaolu
Zhou, Haotan
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12872-023-03648-3
- URN
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urn:nbn:de:101:1-2024022113491277463431
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:00 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Liu, Xin
- Li, Xiaolu
- Zhou, Haotan
- SpringerLink (Online service)
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The malate aspartate shuttle in T cell metabolic fitness: Glutamic oxaloacetic transaminase 1 antagonizes T cell exhaustion
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Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival
The functional role of m6A demethylase ALKBH5 in cardiomyocyte hypertrophy
ADAP1 limits neonatal cardiomyocyte hypertrophy by reducing integrin cell surface expression
LOC102549726/miR-760-3p network is involved in the progression of ISO-induced pathological cardiomyocyte hypertrophy via endoplasmic reticulum stress
MRI based non-invasive detection of cardiomyocyte hypertrophy and cell-volume changes
Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
The malate aspartate shuttle in T cell metabolic fitness: Glutamic oxaloacetic transaminase 1 antagonizes T cell exhaustion
TNIP3 protects against pathological cardiac hypertrophy by stabilizing STAT1
Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival
The functional role of m6A demethylase ALKBH5 in cardiomyocyte hypertrophy
ADAP1 limits neonatal cardiomyocyte hypertrophy by reducing integrin cell surface expression
LOC102549726/miR-760-3p network is involved in the progression of ISO-induced pathological cardiomyocyte hypertrophy via endoplasmic reticulum stress
MRI based non-invasive detection of cardiomyocyte hypertrophy and cell-volume changes
Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
The malate aspartate shuttle in T cell metabolic fitness: Glutamic oxaloacetic transaminase 1 antagonizes T cell exhaustion