The Fanconi anemia core complex promotes CtIP-dependent end resection to drive homologous recombination at DNA double-strand breaks

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: The Fanconi anemia core complex promotes CtIP-dependent end resection to drive homologous recombination at DNA double-strand breaks ; volume:15 ; number:1 ; day:16 ; month:8 ; year:2024 ; pages:1-17 ; date:12.2024
Nature Communications ; 15, Heft 1 (16.8.2024), 1-17, 12.2024

Creator: Kooij, Bert van de
Wal, Fenna J. van der
Rother, Magdalena B.
Wiegant, Wouter W.
Creixell, Pau
Stout, Merula
Joughin, Brian A.
Vornberger, Julia
Altmeyer, Matthias
Vugt, Marcel A. T. M. van
Yaffe, Michael B.
Attikum, Haico van

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41467-024-51090-6

URN: urn:nbn:de:101:1-2411032126598.278783937541

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:35 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Kooij, Bert van de
Wal, Fenna J. van der
Rother, Magdalena B.
Wiegant, Wouter W.
Creixell, Pau
Stout, Merula
Joughin, Brian A.
Vornberger, Julia
Altmeyer, Matthias
Vugt, Marcel A. T. M. van
Yaffe, Michael B.
Attikum, Haico van
SpringerLink (Online service)

Other Objects (12)

Interplay between Fanconi anemia and homologous recombination pathways in genome integrity

Centriolar subdistal appendages promote double‐strand break repair through homologous recombination

Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination

End resection: a key step in homologous recombination and DNA double-strand break repair

The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance

The enrichment of Fanconi anemia/homologous recombination pathway aberrations in ATM/ATR-mutated NSCLC was accompanied by unique molecular features and poor prognosis

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

The Fanconi anemia pathway and ubiquitin

Structural basis of homologous recombination

A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination

Interplay between Fanconi anemia and homologous recombination pathways in genome integrity

Centriolar subdistal appendages promote double‐strand break repair through homologous recombination

Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination

End resection: a key step in homologous recombination and DNA double-strand break repair

The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance

The enrichment of Fanconi anemia/homologous recombination pathway aberrations in ATM/ATR-mutated NSCLC was accompanied by unique molecular features and poor prognosis

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

The Fanconi anemia pathway and ubiquitin

Structural basis of homologous recombination

A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination

Interplay between Fanconi anemia and homologous recombination pathways in genome integrity

Centriolar subdistal appendages promote double‐strand break repair through homologous recombination

Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination

End resection: a key step in homologous recombination and DNA double-strand break repair

The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance

The enrichment of Fanconi anemia/homologous recombination pathway aberrations in ATM/ATR-mutated NSCLC was accompanied by unique molecular features and poor prognosis

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

The Fanconi anemia pathway and ubiquitin

Structural basis of homologous recombination

A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

The Fanconi anemia core complex promotes CtIP-dependent end resection to drive homologous recombination at DNA double-strand breaks

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Interplay between Fanconi anemia and homologous recombination pathways in genome integrity

Centriolar subdistal appendages promote double‐strand break repair through homologous recombination

Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination

End resection: a key step in homologous recombination and DNA double-strand break repair

The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance

The enrichment of Fanconi anemia/homologous recombination pathway aberrations in ATM/ATR-mutated NSCLC was accompanied by unique molecular features and poor prognosis

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

The Fanconi anemia pathway and ubiquitin

Structural basis of homologous recombination

A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination

Interplay between Fanconi anemia and homologous recombination pathways in genome integrity

Centriolar subdistal appendages promote double‐strand break repair through homologous recombination

Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination

End resection: a key step in homologous recombination and DNA double-strand break repair

The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance

The enrichment of Fanconi anemia/homologous recombination pathway aberrations in ATM/ATR-mutated NSCLC was accompanied by unique molecular features and poor prognosis

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

The Fanconi anemia pathway and ubiquitin

Structural basis of homologous recombination

A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination

Interplay between Fanconi anemia and homologous recombination pathways in genome integrity

Centriolar subdistal appendages promote double‐strand break repair through homologous recombination

Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair

One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks

ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination

End resection: a key step in homologous recombination and DNA double-strand break repair

The non-homologous end-joining activity is required for Fanconi anemia fetal HSC maintenance

The enrichment of Fanconi anemia/homologous recombination pathway aberrations in ATM/ATR-mutated NSCLC was accompanied by unique molecular features and poor prognosis

DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

The Fanconi anemia pathway and ubiquitin

Structural basis of homologous recombination

A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination

Related objects

Reset password