End resection: a key step in homologous recombination and DNA double-strand break repair
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2524-7662
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
End resection: a key step in homologous recombination and DNA double-strand break repair ; day:27 ; month:11 ; year:2020 ; pages:1-12
Genome instability & disease ; (27.11.2020), 1-12
- Creator
-
Liu, Sijie
Kong, Daochun
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1007/s42764-020-00028-5
- URN
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urn:nbn:de:101:1-2021010722411197806143
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 15.08.2025, 7:28 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Liu, Sijie
- Kong, Daochun
- SpringerLink (Online service)
Other Objects (12)
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Centriolar subdistal appendages promote double‐strand break repair through homologous recombination
Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair
Dbf4-dependent kinase promotes cell cycle controlled resection of DNA double-strand breaks and repair by homologous recombination
ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
The Fanconi anemia core complex promotes CtIP-dependent end resection to drive homologous recombination at DNA double-strand breaks
The MRE11 nuclease promotes homologous recombination not only in DNA double-strand break resection but also in post-resection in human TK6 cells
A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination
Involvement of the splicing factor SART1 in the BRCA1-dependent homologous recombination repair of DNA double-strand breaks
Effect of Akt and treatment with selenomethionine on homologous recombination-mediated DNA double-strand break repair after ionizing radiation
Publisher Correction: A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Centriolar subdistal appendages promote double‐strand break repair through homologous recombination
Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair
Dbf4-dependent kinase promotes cell cycle controlled resection of DNA double-strand breaks and repair by homologous recombination
ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
The Fanconi anemia core complex promotes CtIP-dependent end resection to drive homologous recombination at DNA double-strand breaks
The MRE11 nuclease promotes homologous recombination not only in DNA double-strand break resection but also in post-resection in human TK6 cells
A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination
Involvement of the splicing factor SART1 in the BRCA1-dependent homologous recombination repair of DNA double-strand breaks
Effect of Akt and treatment with selenomethionine on homologous recombination-mediated DNA double-strand break repair after ionizing radiation
Publisher Correction: A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
Centriolar subdistal appendages promote double‐strand break repair through homologous recombination
Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair
Dbf4-dependent kinase promotes cell cycle controlled resection of DNA double-strand breaks and repair by homologous recombination
ATM Release at Resected Double-Strand Breaks Provides Heterochromatin Reconstitution to Facilitate Homologous Recombination
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
The Fanconi anemia core complex promotes CtIP-dependent end resection to drive homologous recombination at DNA double-strand breaks
The MRE11 nuclease promotes homologous recombination not only in DNA double-strand break resection but also in post-resection in human TK6 cells
A meiosis-specific BRCA2 binding protein recruits recombinases to DNA double-strand breaks to ensure homologous recombination
Involvement of the splicing factor SART1 in the BRCA1-dependent homologous recombination repair of DNA double-strand breaks
Effect of Akt and treatment with selenomethionine on homologous recombination-mediated DNA double-strand break repair after ionizing radiation