- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Göttingen, Georg-August Universität, Diss., 2014
- Classification
-
Medizin, Gesundheit
- Event
-
Veröffentlichung
- (where)
-
Göttingen
- (who)
-
Niedersächsische Staats- und Universitätsbibliothek Göttingen
- (when)
-
2014
- Creator
-
Vasko, Radovan
- Contributor
-
Dihazi, Hassan
Thumm, Michael
- Handle
-
11858/00-1735-0000-0023-995E-C
- URN
-
urn:nbn:de:gbv:7-11858/00-1735-0000-0023-995E-C-7
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:57 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Vasko, Radovan
- Dihazi, Hassan
- Thumm, Michael
- Niedersächsische Staats- und Universitätsbibliothek Göttingen
Time of origin
- 2014
Other Objects (12)
Meta-analyses identify DNA methylation associated with kidney function and damage
Role of Human T Cells in Experimental Hypertension and Hypertensive Kidney Damage
DNA Damage in Chronic Kidney Disease: Evaluation of Clinical Biomarkers
Protein kinase C isoforms in acute kidney injury and chronic kidney disease
Improving flood damage modelling : damage assessment, model development and application
Improving flood damage modelling : damage assessment, model - development and application
Pathological and Biochemical Characterization of Microcystin-Induced Hepatopancreas and Kidney Damage in Carp (Cyprinus carpio)
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
The regulation of nephrin expression in kidney epithelial cells and during inflammatory kidney diseases
Auto-correlation-function-based damage index for damage detection and system identification
Stiff and damage-tolerant metallic glasses
Meta-analyses identify DNA methylation associated with kidney function and damage
Role of Human T Cells in Experimental Hypertension and Hypertensive Kidney Damage
DNA Damage in Chronic Kidney Disease: Evaluation of Clinical Biomarkers
Protein kinase C isoforms in acute kidney injury and chronic kidney disease
Improving flood damage modelling : damage assessment, model development and application
Improving flood damage modelling : damage assessment, model - development and application
Pathological and Biochemical Characterization of Microcystin-Induced Hepatopancreas and Kidney Damage in Carp (Cyprinus carpio)
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
The regulation of nephrin expression in kidney epithelial cells and during inflammatory kidney diseases
Auto-correlation-function-based damage index for damage detection and system identification
Stiff and damage-tolerant metallic glasses
Meta-analyses identify DNA methylation associated with kidney function and damage
Role of Human T Cells in Experimental Hypertension and Hypertensive Kidney Damage
DNA Damage in Chronic Kidney Disease: Evaluation of Clinical Biomarkers
Protein kinase C isoforms in acute kidney injury and chronic kidney disease
Improving flood damage modelling : damage assessment, model development and application
Improving flood damage modelling : damage assessment, model - development and application
Pathological and Biochemical Characterization of Microcystin-Induced Hepatopancreas and Kidney Damage in Carp (Cyprinus carpio)
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
The regulation of nephrin expression in kidney epithelial cells and during inflammatory kidney diseases
Auto-correlation-function-based damage index for damage detection and system identification