Hochschulschrift
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
Heidelberg, Universität Heidelberg, Dissertation, 2018
- Klassifikation
-
Naturwissenschaften
- Schlagwort
-
Diabetes
Glucose
Kidneys--Diseases
Diabetes mellitus
Mitochondrium
Nephropathia diabetica
- Ereignis
-
Veröffentlichung
- (wo)
-
Heidelberg
- (wer)
-
Universitätsbibliothek Heidelberg
- (wann)
-
2018
- Urheber
- Beteiligte Personen und Organisationen
- DOI
-
10.11588/heidok.00024049
- URN
-
urn:nbn:de:bsz:16-heidok-240491
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:54 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Objekttyp
- Hochschulschrift
Beteiligte
- Volk, Nadine
- Nawroth, Peter Paul
- Universitätsbibliothek Heidelberg
Entstanden
- 2018
Ähnliche Objekte (12)
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
Scutellarin Rescued Mitochondrial Damage through Ameliorating Mitochondrial Glucose Oxidation via the Pdk‐Pdc Axis
Fluvastatin-induced myofibrillar damage is associated with elevated ROS, and impaired fatty acid oxidation, and is preceded by mitochondrial morphological changes
Novel meriolin derivatives activate the mitochondrial apoptosis pathway in the presence of antiapoptotic Bcl-2
Activate your inner healer
Mitochondrial dysfunction and oxidative damage in neurodegenerative diseases
Mitochondrial damage and axon degeneration in chronic neuroinflammation
Elevated CHCHD4 orchestrates mitochondrial oxidative phosphorylation to disturb hypoxic pulmonary hypertension
The GLP-1 receptor agonists exenatide and liraglutide activate Glucose transport by an AMPK-dependent mechanism
Alpha-synuclein suppresses mitochondrial protease ClpP to trigger mitochondrial oxidative damage and neurotoxicity
Metabolic memory in mitochondrial oxidative damage triggers diabetic retinopathy
G2019S selective LRRK2 kinase inhibitor abrogates mitochondrial DNA damage
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
Scutellarin Rescued Mitochondrial Damage through Ameliorating Mitochondrial Glucose Oxidation via the Pdk‐Pdc Axis
Fluvastatin-induced myofibrillar damage is associated with elevated ROS, and impaired fatty acid oxidation, and is preceded by mitochondrial morphological changes
Novel meriolin derivatives activate the mitochondrial apoptosis pathway in the presence of antiapoptotic Bcl-2
Activate your inner healer
Mitochondrial dysfunction and oxidative damage in neurodegenerative diseases
Mitochondrial damage and axon degeneration in chronic neuroinflammation
Elevated CHCHD4 orchestrates mitochondrial oxidative phosphorylation to disturb hypoxic pulmonary hypertension
The GLP-1 receptor agonists exenatide and liraglutide activate Glucose transport by an AMPK-dependent mechanism
Alpha-synuclein suppresses mitochondrial protease ClpP to trigger mitochondrial oxidative damage and neurotoxicity
Metabolic memory in mitochondrial oxidative damage triggers diabetic retinopathy
G2019S selective LRRK2 kinase inhibitor abrogates mitochondrial DNA damage
Diabetes-induced mitochondrial changes are tissue- and model-dependent and elevated glucose levels do not activate the pathways of hyperglycemic damage in the kidney
Scutellarin Rescued Mitochondrial Damage through Ameliorating Mitochondrial Glucose Oxidation via the Pdk‐Pdc Axis
Fluvastatin-induced myofibrillar damage is associated with elevated ROS, and impaired fatty acid oxidation, and is preceded by mitochondrial morphological changes
Novel meriolin derivatives activate the mitochondrial apoptosis pathway in the presence of antiapoptotic Bcl-2
Activate your inner healer
Mitochondrial dysfunction and oxidative damage in neurodegenerative diseases
Mitochondrial damage and axon degeneration in chronic neuroinflammation
Elevated CHCHD4 orchestrates mitochondrial oxidative phosphorylation to disturb hypoxic pulmonary hypertension
The GLP-1 receptor agonists exenatide and liraglutide activate Glucose transport by an AMPK-dependent mechanism
Alpha-synuclein suppresses mitochondrial protease ClpP to trigger mitochondrial oxidative damage and neurotoxicity
Metabolic memory in mitochondrial oxidative damage triggers diabetic retinopathy