Sphingomyelin synthase 2 promotes the stemness of breast cancer cells via modulating NF-κB signaling pathway
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Sphingomyelin synthase 2 promotes the stemness of breast cancer cells via modulating NF-κB signaling pathway ; volume:150 ; number:2 ; day:29 ; month:1 ; year:2024 ; pages:1-12 ; date:2.2024
Journal of cancer research and clinical oncology ; 150, Heft 2 (29.1.2024), 1-12, 2.2024
- Creator
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Feng, Haizhan
Dong, Yahui
Chen, Kunling
You, Zicong
Weng, Junyan
Liang, Peiqiao
Shi, Fujun
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s00432-023-05589-y
- URN
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urn:nbn:de:101:1-2405232131487.115059450774
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 11:03 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Feng, Haizhan
- Dong, Yahui
- Chen, Kunling
- You, Zicong
- Weng, Junyan
- Liang, Peiqiao
- Shi, Fujun
- SpringerLink (Online service)
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Deficiency of sphingomyelin synthase 1 but not sphingomyelin synthase 2 reduces bone formation due to impaired osteoblast differentiation
Subcellular Targeting Domains of Sphingomyelin Synthase 1 and 2
Sphingomyelin synthase 2 promotes an aggressive breast cancer phenotype by disrupting the homoeostasis of ceramide and sphingomyelin
The biochemical, immunological and molecular characterization of human sphingomyelin synthase-1
Biological functions of Sphingomyelin synthase related protein and Ceramide synthase 4 investigated with transgenic mouse mutants
Role of collagen degradation pathway in sphingomyelin synthase 2-deficient mouse skin
Adenovirus-mediated sphingomyelin synthase 2 increases atherosclerotic lesions in ApoE KO mice
Sphingomyelin synthase overexpression increases cholesterol accumulation and decreases cholesterol secretion in liver cells
Interaction of NF-κB and FOSL1 drives glioma stemness
The role of Sphingomyelin synthase 2 (SMS2) in platelet activation and its clinical significance
Exploring the physiological roles of sphingomyelin synthase family members by proteolytic inactivation and mistargeting strategies
Farnesyl diphosphate synthase is important for the maintenance of glioblastoma stemness
Deficiency of sphingomyelin synthase 1 but not sphingomyelin synthase 2 reduces bone formation due to impaired osteoblast differentiation
Subcellular Targeting Domains of Sphingomyelin Synthase 1 and 2
Sphingomyelin synthase 2 promotes an aggressive breast cancer phenotype by disrupting the homoeostasis of ceramide and sphingomyelin
The biochemical, immunological and molecular characterization of human sphingomyelin synthase-1
Biological functions of Sphingomyelin synthase related protein and Ceramide synthase 4 investigated with transgenic mouse mutants
Role of collagen degradation pathway in sphingomyelin synthase 2-deficient mouse skin
Adenovirus-mediated sphingomyelin synthase 2 increases atherosclerotic lesions in ApoE KO mice
Sphingomyelin synthase overexpression increases cholesterol accumulation and decreases cholesterol secretion in liver cells
Interaction of NF-κB and FOSL1 drives glioma stemness
The role of Sphingomyelin synthase 2 (SMS2) in platelet activation and its clinical significance
Exploring the physiological roles of sphingomyelin synthase family members by proteolytic inactivation and mistargeting strategies