Hochschulschrift
Exploring the physiological roles of sphingomyelin synthase family members by proteolytic inactivation and mistargeting strategies
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Dimensions
-
30 cm
- Extent
-
V, 106 Blätter
- Language
-
Englisch
- Notes
-
Illustrationen, Diagramme
University of Osnabrück, Dissertation, 2019
- Classification
-
Biowissenschaften, Biologie
- Event
-
Veröffentlichung
- (where)
-
Osnabrück
- (when)
-
September 2019
- Creator
-
Parolek, Jan
- Table of contents
- Rights
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Last update
-
11.06.2025, 2:06 PM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Parolek, Jan
Time of origin
- September 2019
Other Objects (12)
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Subcellular Targeting Domains of Sphingomyelin Synthase 1 and 2
Sphingomyelin synthase 2 promotes an aggressive breast cancer phenotype by disrupting the homoeostasis of ceramide and sphingomyelin
The biochemical, immunological and molecular characterization of human sphingomyelin synthase-1
Biological functions of Sphingomyelin synthase related protein and Ceramide synthase 4 investigated with transgenic mouse mutants
Role of collagen degradation pathway in sphingomyelin synthase 2-deficient mouse skin
Proteolytic activation of fatty acid synthase signals pan-stress resolution
Adenovirus-mediated sphingomyelin synthase 2 increases atherosclerotic lesions in ApoE KO mice
Sphingomyelin synthase overexpression increases cholesterol accumulation and decreases cholesterol secretion in liver cells
The role of Sphingomyelin synthase 2 (SMS2) in platelet activation and its clinical significance
Mechanism of glycogen synthase inactivation and interaction with glycogenin
Heme-dependent Inactivation of 5-Aminolevulinate Synthase from Caulobacter crescentus
Deficiency of sphingomyelin synthase 1 but not sphingomyelin synthase 2 reduces bone formation due to impaired osteoblast differentiation
Subcellular Targeting Domains of Sphingomyelin Synthase 1 and 2
Sphingomyelin synthase 2 promotes an aggressive breast cancer phenotype by disrupting the homoeostasis of ceramide and sphingomyelin
The biochemical, immunological and molecular characterization of human sphingomyelin synthase-1
Biological functions of Sphingomyelin synthase related protein and Ceramide synthase 4 investigated with transgenic mouse mutants
Role of collagen degradation pathway in sphingomyelin synthase 2-deficient mouse skin
Proteolytic activation of fatty acid synthase signals pan-stress resolution
Adenovirus-mediated sphingomyelin synthase 2 increases atherosclerotic lesions in ApoE KO mice
Sphingomyelin synthase overexpression increases cholesterol accumulation and decreases cholesterol secretion in liver cells
The role of Sphingomyelin synthase 2 (SMS2) in platelet activation and its clinical significance
Mechanism of glycogen synthase inactivation and interaction with glycogenin
Heme-dependent Inactivation of 5-Aminolevulinate Synthase from Caulobacter crescentus
Deficiency of sphingomyelin synthase 1 but not sphingomyelin synthase 2 reduces bone formation due to impaired osteoblast differentiation
Subcellular Targeting Domains of Sphingomyelin Synthase 1 and 2
Sphingomyelin synthase 2 promotes an aggressive breast cancer phenotype by disrupting the homoeostasis of ceramide and sphingomyelin
The biochemical, immunological and molecular characterization of human sphingomyelin synthase-1
Biological functions of Sphingomyelin synthase related protein and Ceramide synthase 4 investigated with transgenic mouse mutants
Role of collagen degradation pathway in sphingomyelin synthase 2-deficient mouse skin
Proteolytic activation of fatty acid synthase signals pan-stress resolution
Adenovirus-mediated sphingomyelin synthase 2 increases atherosclerotic lesions in ApoE KO mice
Sphingomyelin synthase overexpression increases cholesterol accumulation and decreases cholesterol secretion in liver cells
The role of Sphingomyelin synthase 2 (SMS2) in platelet activation and its clinical significance
Mechanism of glycogen synthase inactivation and interaction with glycogenin