- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2051-5960
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
ALS-linked misfolded SOD1 species have divergent impacts on mitochondria ; volume:4 ; number:1 ; day:27 ; month:4 ; year:2016 ; pages:1-14 ; date:12.2016
Acta Neuropathologica Communications ; 4, Heft 1 (27.4.2016), 1-14, 12.2016
- Creator
-
Pickles, Sarah
- Contributor
-
Semmler, Sabrina
Broom, Helen R.
Destroismaisons, Laurie
Legroux, Laurine
Arbour, Nathalie
Meiering, Elizabeth
Cashman, Neil R.
Vande Velde, Christine
SpringerLink (Online service)
- DOI
-
10.1186/s40478-016-0313-8
- URN
-
urn:nbn:de:1111-2016042815424
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:47 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Pickles, Sarah
- Semmler, Sabrina
- Broom, Helen R.
- Destroismaisons, Laurie
- Legroux, Laurine
- Arbour, Nathalie
- Meiering, Elizabeth
- Cashman, Neil R.
- Vande Velde, Christine
- SpringerLink (Online service)
Other Objects (12)
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Spinal cord abnormal autophagy and mitochondria energy metabolism are modified by swim training in SOD1-G93A mice
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
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SIRT1 overexpression ameliorates a mouse model of SOD1-linked amyotrophic lateral sclerosis via HSF1/HSP70i chaperone system
Features of wild-type human SOD1 limit interactions with misfolded aggregates of mouse G86R Sod1
Divergent
Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history
Increased surface P2X4 receptors by mutant SOD1 proteins contribute to ALS pathogenesis in SOD1-G93A mice
Mitochondrial damage revealed by immunoselection for ALS-linked misfolded SOD1
K63-linked ubiquitylation induces global sequestration of mitochondria
Extreme diversity of 12 cations in folding ALS-linked hSOD1 unveils novel hSOD1-dependent mechanisms for Fe2+/Cu2+-induced cytotoxicity
Spinal cord abnormal autophagy and mitochondria energy metabolism are modified by swim training in SOD1-G93A mice
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Die Beteiligung von wildtypischer SOD1 an der Pathogenese der SOD1-vermittelten amyotrophen Lateralsklerose (ALS1)
SIRT1 overexpression ameliorates a mouse model of SOD1-linked amyotrophic lateral sclerosis via HSF1/HSP70i chaperone system
Features of wild-type human SOD1 limit interactions with misfolded aggregates of mouse G86R Sod1
Divergent
Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history
Increased surface P2X4 receptors by mutant SOD1 proteins contribute to ALS pathogenesis in SOD1-G93A mice
Mitochondrial damage revealed by immunoselection for ALS-linked misfolded SOD1
K63-linked ubiquitylation induces global sequestration of mitochondria
Extreme diversity of 12 cations in folding ALS-linked hSOD1 unveils novel hSOD1-dependent mechanisms for Fe2+/Cu2+-induced cytotoxicity
Spinal cord abnormal autophagy and mitochondria energy metabolism are modified by swim training in SOD1-G93A mice
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Die Beteiligung von wildtypischer SOD1 an der Pathogenese der SOD1-vermittelten amyotrophen Lateralsklerose (ALS1)
SIRT1 overexpression ameliorates a mouse model of SOD1-linked amyotrophic lateral sclerosis via HSF1/HSP70i chaperone system
Features of wild-type human SOD1 limit interactions with misfolded aggregates of mouse G86R Sod1
Divergent
Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history