- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1750-1326
- Extent
-
Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Mitochondrial damage revealed by immunoselection for ALS-linked misfolded SOD1 ; volume:8 ; number:1 ; day:4 ; month:10 ; year:2013 ; pages:1-2 ; date:9.2013
Molecular neurodegeneration ; 8, Heft 1 (4.10.2013), 1-2, 9.2013
- Creator
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Pickles, Sarah
- Contributor
-
Destroismaisons, Laurie
Peyrard, Sarah
Cadot, Sarah
Rouleau, Guy
Brown, Robert
Julien, Jean-Pierre
Arbour, Nathalie
Velde, Christine Vande
SpringerLink (Online service)
- DOI
-
10.1186/1750-1326-8-S1-P70
- URN
-
urn:nbn:de:1111-2016073072625
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:52 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Pickles, Sarah
- Destroismaisons, Laurie
- Peyrard, Sarah
- Cadot, Sarah
- Rouleau, Guy
- Brown, Robert
- Julien, Jean-Pierre
- Arbour, Nathalie
- Velde, Christine Vande
- SpringerLink (Online service)
Other Objects (12)
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SIRT1 overexpression ameliorates a mouse model of SOD1-linked amyotrophic lateral sclerosis via HSF1/HSP70i chaperone system
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Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history
Increased surface P2X4 receptors by mutant SOD1 proteins contribute to ALS pathogenesis in SOD1-G93A mice
ALS-linked misfolded SOD1 species have divergent impacts on mitochondria
Extreme diversity of 12 cations in folding ALS-linked hSOD1 unveils novel hSOD1-dependent mechanisms for Fe2+/Cu2+-induced cytotoxicity
Herpes simplex encephalitis is linked with selective mitochondrial damage; a post-mortem and in vitro study
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Impairment of mitochondrial calcium handling in a mtSOD1 cell culture model of motoneuron disease
Die Beteiligung von wildtypischer SOD1 an der Pathogenese der SOD1-vermittelten amyotrophen Lateralsklerose (ALS1)
SIRT1 overexpression ameliorates a mouse model of SOD1-linked amyotrophic lateral sclerosis via HSF1/HSP70i chaperone system
Features of wild-type human SOD1 limit interactions with misfolded aggregates of mouse G86R Sod1
Skeletal myotubes expressing ALS mutant SOD1 induce pathogenic changes, impair mitochondrial axonal transport, and trigger motoneuron death
Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history
Increased surface P2X4 receptors by mutant SOD1 proteins contribute to ALS pathogenesis in SOD1-G93A mice
ALS-linked misfolded SOD1 species have divergent impacts on mitochondria
Extreme diversity of 12 cations in folding ALS-linked hSOD1 unveils novel hSOD1-dependent mechanisms for Fe2+/Cu2+-induced cytotoxicity
Herpes simplex encephalitis is linked with selective mitochondrial damage; a post-mortem and in vitro study
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Impairment of mitochondrial calcium handling in a mtSOD1 cell culture model of motoneuron disease
Die Beteiligung von wildtypischer SOD1 an der Pathogenese der SOD1-vermittelten amyotrophen Lateralsklerose (ALS1)
SIRT1 overexpression ameliorates a mouse model of SOD1-linked amyotrophic lateral sclerosis via HSF1/HSP70i chaperone system
Features of wild-type human SOD1 limit interactions with misfolded aggregates of mouse G86R Sod1
Skeletal myotubes expressing ALS mutant SOD1 induce pathogenic changes, impair mitochondrial axonal transport, and trigger motoneuron death
Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history