Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1744-8069
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain ; volume:6 ; number:1 ; day:10 ; month:12 ; year:2010 ; pages:1-14 ; date:12.2010
Molecular pain ; 6, Heft 1 (10.12.2010), 1-14, 12.2010
- Creator
-
Serrano, Alexandre
Paré, Michel
McIntosh, Fraser
Elmes, Steven JR
Martino, Giovanni
Jomphe, Claudia
Lessard, Etienne
Lembo, Paola MC
Vaillancourt, François
Perkins, Martin N.
Cao, Chang Qing
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/1744-8069-6-90
- URN
-
urn:nbn:de:101:1-2021090121002637519388
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:27 AM CEST
Data provider
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Associated
- Serrano, Alexandre
- Paré, Michel
- McIntosh, Fraser
- Elmes, Steven JR
- Martino, Giovanni
- Jomphe, Claudia
- Lessard, Etienne
- Lembo, Paola MC
- Vaillancourt, François
- Perkins, Martin N.
- Cao, Chang Qing
- SpringerLink (Online service)
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The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
Chemokine CCL2 and its receptor CCR2 in the medullary dorsal horn are involved in trigeminal neuropathic pain
The GlyT1 inhibitor bitopertin ameliorates Allodynia and Hyperalgesia in animal models of neuropathic and inflammatory pain
Lack of mechanical and thermal allodynia, and thermal hyperalgesia induced by peripheral neuropathic pain in NOS2 knockout mice
Coexistence of chronic hyperalgesia and multilevel neuroinflammatory responses after experimental SCI: a systematic approach to profiling neuropathic pain
Targeted mutation of EphB1 receptor prevents development of neuropathic hyperalgesia and physical dependence on morphine in mice
Upregulation of the hypothalamo-neurohypophysial system and activation of vasopressin neurones attenuates hyperalgesia in a neuropathic pain model rat
Blocking Mammalian Target of Rapamycin (mTOR) Alleviates Neuropathic Pain Induced by Chemotherapeutic Bortezomib
Sub-paresthesia spinal cord stimulation reverses thermal hyperalgesia and modulates low frequency EEG in a rat model of neuropathic pain
Targeted blocking of CCR2 and CXCR2 improves the efficacy of transarterial chemoembolization of hepatocarcinoma
Blocking TRPA1 and TNF-α Signal Improves Bortezomib-Induced Neuropathic Pain
The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
Chemokine CCL2 and its receptor CCR2 in the medullary dorsal horn are involved in trigeminal neuropathic pain
The GlyT1 inhibitor bitopertin ameliorates Allodynia and Hyperalgesia in animal models of neuropathic and inflammatory pain
Lack of mechanical and thermal allodynia, and thermal hyperalgesia induced by peripheral neuropathic pain in NOS2 knockout mice
Coexistence of chronic hyperalgesia and multilevel neuroinflammatory responses after experimental SCI: a systematic approach to profiling neuropathic pain
Targeted mutation of EphB1 receptor prevents development of neuropathic hyperalgesia and physical dependence on morphine in mice
Upregulation of the hypothalamo-neurohypophysial system and activation of vasopressin neurones attenuates hyperalgesia in a neuropathic pain model rat
Blocking Mammalian Target of Rapamycin (mTOR) Alleviates Neuropathic Pain Induced by Chemotherapeutic Bortezomib