Targeted mutation of EphB1 receptor prevents development of neuropathic hyperalgesia and physical dependence on morphine in mice
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1744-8069
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Targeted mutation of EphB1 receptor prevents development of neuropathic hyperalgesia and physical dependence on morphine in mice ; volume:4 ; number:1 ; day:21 ; month:11 ; year:2008 ; pages:1-12 ; date:12.2008
Molecular pain ; 4, Heft 1 (21.11.2008), 1-12, 12.2008
- Urheber
-
Han, Yuan
Song, Xue-Song
Liu, Wen-Tao
Henkemeyer, Mark
Song, Xue-Jun
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/1744-8069-4-60
- URN
-
urn:nbn:de:101:1-2021082919540642358611
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:31 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Han, Yuan
- Song, Xue-Song
- Liu, Wen-Tao
- Henkemeyer, Mark
- Song, Xue-Jun
- SpringerLink (Online service)
Ähnliche Objekte (12)
Pre-injury administration of morphine prevents development of neuropathic hyperalgesia through activation of descending monoaminergic mechanisms in the spinal cord in mice
Morphine-induced hyperalgesia involves mu opioid receptors and the metabolite morphine-3-glucuronide
Melatonin prevents morphine-induced hyperalgesia and tolerance in rats: role of protein kinase C and N-methyl-D-aspartate receptors
Pharmacological blockade of neurokinin1 receptor restricts morphine-induced tolerance and hyperalgesia in the rat
Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain
Dextromethorphan attenuated the higher vulnerability to inflammatory thermal hyperalgesia caused by prenatal morphine exposure in rat offspring
Short-chain fatty acid, butyrate prevents morphine-and paclitaxel-induced nociceptive hypersensitivity
The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
Morphine
The YTHDF1–TRAF6 pathway regulates the neuroinflammatory response and contributes to morphine tolerance and hyperalgesia in the periaqueductal gray
The GlyT1 inhibitor bitopertin ameliorates Allodynia and Hyperalgesia in animal models of neuropathic and inflammatory pain
Pre-injury administration of morphine prevents development of neuropathic hyperalgesia through activation of descending monoaminergic mechanisms in the spinal cord in mice
Morphine-induced hyperalgesia involves mu opioid receptors and the metabolite morphine-3-glucuronide
Melatonin prevents morphine-induced hyperalgesia and tolerance in rats: role of protein kinase C and N-methyl-D-aspartate receptors
Pharmacological blockade of neurokinin1 receptor restricts morphine-induced tolerance and hyperalgesia in the rat
Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain
Dextromethorphan attenuated the higher vulnerability to inflammatory thermal hyperalgesia caused by prenatal morphine exposure in rat offspring
Short-chain fatty acid, butyrate prevents morphine-and paclitaxel-induced nociceptive hypersensitivity
The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
Morphine
The YTHDF1–TRAF6 pathway regulates the neuroinflammatory response and contributes to morphine tolerance and hyperalgesia in the periaqueductal gray
The GlyT1 inhibitor bitopertin ameliorates Allodynia and Hyperalgesia in animal models of neuropathic and inflammatory pain
Pre-injury administration of morphine prevents development of neuropathic hyperalgesia through activation of descending monoaminergic mechanisms in the spinal cord in mice
Morphine-induced hyperalgesia involves mu opioid receptors and the metabolite morphine-3-glucuronide
Melatonin prevents morphine-induced hyperalgesia and tolerance in rats: role of protein kinase C and N-methyl-D-aspartate receptors
Pharmacological blockade of neurokinin1 receptor restricts morphine-induced tolerance and hyperalgesia in the rat
Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain
Dextromethorphan attenuated the higher vulnerability to inflammatory thermal hyperalgesia caused by prenatal morphine exposure in rat offspring
Short-chain fatty acid, butyrate prevents morphine-and paclitaxel-induced nociceptive hypersensitivity
The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
Morphine
The YTHDF1–TRAF6 pathway regulates the neuroinflammatory response and contributes to morphine tolerance and hyperalgesia in the periaqueductal gray