PDEδ interference impedes the proliferation and survival of oncogenic KRas expressing human cancer cell lines
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Dortmund, Technische Universität, Dissertation, 2019
- Keyword
-
Cancer
Cell lines
Survival
Krebs
Medizin
- Event
-
Veröffentlichung
- (where)
-
Dortmund
- (who)
-
Universitätsbibliothek Dortmund
- (when)
-
2018
- Creator
- Contributor
-
Bastiaens, Philippe
Dehmelt, Leif
- Handle
-
2003/38045
- URN
-
urn:nbn:de:101:1-2019051703422201162960
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.03.2024, 11:43 AM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Klein, Christian H.
- Bastiaens, Philippe
- Dehmelt, Leif
- Universitätsbibliothek Dortmund
Time of origin
- 2018
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Oncogenic KRAS renders cells resistant to ferroptosis by upregulating FSP1
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HDAC Inhibitor-Induced acetylation of KRAS confers a pro-oncogenic effect limiting treatment response in colorectal cancer
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Oncogenic KrasG12D causes myeloproliferation via NLRP3 inflammasome activation
Oncogenic KRAS supports pancreatic cancer through regulation of nucleotide synthesis
Oncogenic KRAS renders cells resistant to ferroptosis by upregulating FSP1
Oncogenic KrasG12D causes myeloproliferation in leukaemia via NLRP3 inflammasome activation
The role of oncogenic Kras signaling in the non-hematopoietic bone marrow microenvironment
"KRAS
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Cell type-dependent differential activation of ERK by oncogenic KRAS or BRAF in the mouse intestinal epithelium
HDAC Inhibitor-Induced acetylation of KRAS confers a pro-oncogenic effect limiting treatment response in colorectal cancer
Downstream Signaling of Oncogenic Kras in Pancreatic Ductal Adenocarcinoma : In Vivo and Ex Vivo Imaging of Tumor Cell Proliferation as a Tool for the Identification of Therapeutic Targets