Radiotherapy-induced cell death activates paracrine HMGB1-TLR2 signaling and accelerates pancreatic carcinoma metastasis

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1756-9966

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Radiotherapy-induced cell death activates paracrine HMGB1-TLR2 signaling and accelerates pancreatic carcinoma metastasis ; volume:37 ; number:1 ; day:3 ; month:4 ; year:2018 ; pages:1-15 ; date:12.2018
Journal of experimental & clinical cancer research ; 37, Heft 1 (3.4.2018), 1-15, 12.2018

Creator: Chen, Xuelian

Contributor: Zhang, Lirong
Jiang, Yujie
Song, Lian
Liu, Yanfang
Cheng, Fang
Fan, Xin
Cao, Xiongfeng
Gong, Aihua
Wang, Dongqing
Zhu, Haitao
SpringerLink (Online service)

DOI: 10.1186/s13046-018-0726-2

URN: urn:nbn:de:101:1-2018060921525151094483

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:33 AM CEST

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Associated

Chen, Xuelian
Zhang, Lirong
Jiang, Yujie
Song, Lian
Liu, Yanfang
Cheng, Fang
Fan, Xin
Cao, Xiongfeng
Gong, Aihua
Wang, Dongqing
Zhu, Haitao
SpringerLink (Online service)

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Radiotherapy-induced cell death activates paracrine HMGB1-TLR2 signaling and accelerates pancreatic carcinoma metastasis

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Other Objects (12)

HMGB1 released by irradiated tumor cells promotes living tumor cell proliferation via paracrine effect

Aggravated renal fibrosis is positively associated with the activation of HMGB1-TLR2/4 signaling in STZ-induced diabetic mice

V1bR enhances glucose-stimulated insulin secretion by paracrine production of glucagon which activates GLP-1 receptor

Generation of anti-TLR2 intrabody mediating inhibition of macrophage surface TLR2 expression and TLR2-driven cell activation

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V1bR enhances glucose-stimulated insulin secretion by paracrine production of glucagon which activates GLP-1 receptor

Generation of anti-TLR2 intrabody mediating inhibition of macrophage surface TLR2 expression and TLR2-driven cell activation

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Aggravated renal fibrosis is positively associated with the activation of HMGB1-TLR2/4 signaling in STZ-induced diabetic mice

V1bR enhances glucose-stimulated insulin secretion by paracrine production of glucagon which activates GLP-1 receptor

Generation of anti-TLR2 intrabody mediating inhibition of macrophage surface TLR2 expression and TLR2-driven cell activation

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