Inhibition of HMGB1/RAGE-mediated endocytosis by HMGB1 antagonist box A, anti-HMGB1 antibodies, and cholinergic agonists suppresses inflammation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1528-3658
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Inhibition of HMGB1/RAGE-mediated endocytosis by HMGB1 antagonist box A, anti-HMGB1 antibodies, and cholinergic agonists suppresses inflammation ; volume:25 ; number:1 ; day:11 ; month:4 ; year:2019 ; pages:1-13 ; date:12.2019
Molecular medicine ; 25, Heft 1 (11.4.2019), 1-13, 12.2019
- Creator
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Yang, Huan
Liu, Hui
Zeng, Qiong
Imperato, Gavin H.
Addorisio, Meghan E.
Li, Jianhua
He, Mingzhu
Cheng, Kai Fan
Al-Abed, Yousef
Harris, Helena E.
Chavan, Sangeeta S.
Andersson, Ulf
Tracey, Kevin J.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s10020-019-0081-6
- URN
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urn:nbn:de:101:1-2019042923505442383357
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:47 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Yang, Huan
- Liu, Hui
- Zeng, Qiong
- Imperato, Gavin H.
- Addorisio, Meghan E.
- Li, Jianhua
- He, Mingzhu
- Cheng, Kai Fan
- Al-Abed, Yousef
- Harris, Helena E.
- Chavan, Sangeeta S.
- Andersson, Ulf
- Tracey, Kevin J.
- SpringerLink (Online service)
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Redox modifications of cysteine residues regulate the cytokine activity of HMGB1
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HMGB1, sRAGE und Nukleosomen als frühzeitige Prognosemarker bei polytraumatisierten Patienten
Mechanism of HMGB1–RAGE in Kawasaki disease with coronary artery injury
Untersuchungen zur Rolle der HMGB1/RAGE-Achse bei hepatischer Nekroentzündung in Zellkultur
Extracellular HMGB1 interacts with RAGE and promotes chemoresistance in acute leukemia cells
Ethyl pyruvate reduces organic dust-induced airway inflammation by targeting HMGB1-RAGE signaling
Retraction Note to: Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)
Expression of Concern to: Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)
HMGB1 promotes ERK-mediated mitochondrial Drp1 phosphorylation for chemoresistance through RAGE in colorectal cancer
Fas (CD95) induces rapid, TLR4/IRAK4-dependent release of pro-inflammatory HMGB1 from macrophages
Redox modifications of cysteine residues regulate the cytokine activity of HMGB1
HMGB1 in inflammation and cancer
HMGB1, neuronal excitability and epilepsy
HMGB1, sRAGE und Nukleosomen als frühzeitige Prognosemarker bei polytraumatisierten Patienten
Mechanism of HMGB1–RAGE in Kawasaki disease with coronary artery injury
Untersuchungen zur Rolle der HMGB1/RAGE-Achse bei hepatischer Nekroentzündung in Zellkultur
Extracellular HMGB1 interacts with RAGE and promotes chemoresistance in acute leukemia cells
Ethyl pyruvate reduces organic dust-induced airway inflammation by targeting HMGB1-RAGE signaling
Retraction Note to: Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)
Expression of Concern to: Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)
HMGB1 promotes ERK-mediated mitochondrial Drp1 phosphorylation for chemoresistance through RAGE in colorectal cancer
Fas (CD95) induces rapid, TLR4/IRAK4-dependent release of pro-inflammatory HMGB1 from macrophages
Redox modifications of cysteine residues regulate the cytokine activity of HMGB1
HMGB1 in inflammation and cancer
HMGB1, neuronal excitability and epilepsy
HMGB1, sRAGE und Nukleosomen als frühzeitige Prognosemarker bei polytraumatisierten Patienten
Mechanism of HMGB1–RAGE in Kawasaki disease with coronary artery injury
Untersuchungen zur Rolle der HMGB1/RAGE-Achse bei hepatischer Nekroentzündung in Zellkultur
Extracellular HMGB1 interacts with RAGE and promotes chemoresistance in acute leukemia cells
Ethyl pyruvate reduces organic dust-induced airway inflammation by targeting HMGB1-RAGE signaling
Retraction Note to: Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)
Expression of Concern to: Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)