FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1465-542X

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors ; volume:22 ; number:1 ; day:3 ; month:6 ; year:2020 ; pages:1-15 ; date:12.2020
Breast cancer research ; 22, Heft 1 (3.6.2020), 1-15, 12.2020

Creator: Paul, Ritama
Luo, Ming
Mo, Xueying
Lu, Jason
Yeo, Syn Kok
Guan, Jun-Lin

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13058-020-01298-3

URN: urn:nbn:de:101:1-2020083020034127446612

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:01 AM CEST

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Paul, Ritama
Luo, Ming
Mo, Xueying
Lu, Jason
Yeo, Syn Kok
Guan, Jun-Lin
SpringerLink (Online service)

Other Objects (12)

Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers

Human basal-like breast cancer is represented by one of the two mammary tumor subtypes in dogs

Weight loss reduces basal-like breast cancer through kinome reprogramming

Targeted Pten deletion plus p53-R270H mutation in mouse mammary epithelium induces aggressive claudin-low and basal-like breast cancer

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

The CD44+/CD24-phenotype is enriched in basal-like breast tumors

Gene expression signatures of morphologically normal breast tissue identify basal-like tumors

Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers

Human basal-like breast cancer is represented by one of the two mammary tumor subtypes in dogs

Weight loss reduces basal-like breast cancer through kinome reprogramming

Targeted Pten deletion plus p53-R270H mutation in mouse mammary epithelium induces aggressive claudin-low and basal-like breast cancer

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

The CD44+/CD24-phenotype is enriched in basal-like breast tumors

Gene expression signatures of morphologically normal breast tissue identify basal-like tumors

Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers

Human basal-like breast cancer is represented by one of the two mammary tumor subtypes in dogs

Weight loss reduces basal-like breast cancer through kinome reprogramming

Targeted Pten deletion plus p53-R270H mutation in mouse mammary epithelium induces aggressive claudin-low and basal-like breast cancer

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

The CD44+/CD24-phenotype is enriched in basal-like breast tumors

Gene expression signatures of morphologically normal breast tissue identify basal-like tumors

Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

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FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors

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Other Objects (12)

Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers

Human basal-like breast cancer is represented by one of the two mammary tumor subtypes in dogs

Weight loss reduces basal-like breast cancer through kinome reprogramming

Targeted Pten deletion plus p53-R270H mutation in mouse mammary epithelium induces aggressive claudin-low and basal-like breast cancer

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

The CD44+/CD24-phenotype is enriched in basal-like breast tumors

Gene expression signatures of morphologically normal breast tissue identify basal-like tumors

Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers

Human basal-like breast cancer is represented by one of the two mammary tumor subtypes in dogs

Weight loss reduces basal-like breast cancer through kinome reprogramming

Targeted Pten deletion plus p53-R270H mutation in mouse mammary epithelium induces aggressive claudin-low and basal-like breast cancer

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

The CD44+/CD24-phenotype is enriched in basal-like breast tumors

Gene expression signatures of morphologically normal breast tissue identify basal-like tumors

Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers

Human basal-like breast cancer is represented by one of the two mammary tumor subtypes in dogs

Weight loss reduces basal-like breast cancer through kinome reprogramming

Targeted Pten deletion plus p53-R270H mutation in mouse mammary epithelium induces aggressive claudin-low and basal-like breast cancer

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

The CD44+/CD24-phenotype is enriched in basal-like breast tumors

Gene expression signatures of morphologically normal breast tissue identify basal-like tumors

Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

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