The CD44+/CD24-phenotype is enriched in basal-like breast tumors

zu Verbundenen Objekten

Standort
Deutsche Nationalbibliothek Frankfurt am Main
ISSN
1465-542X
Umfang
Online-Ressource
Sprache
Englisch
Anmerkungen
online resource.

Erschienen in
The CD44+/CD24-phenotype is enriched in basal-like breast tumors ; volume:10 ; number:3 ; day:17 ; month:6 ; year:2008 ; pages:1-12 ; date:6.2008
Breast cancer research ; 10, Heft 3 (17.6.2008), 1-12, 6.2008

Urheber
Honeth, Gabriella
Beteiligte Personen und Organisationen
Bendahl, Pär-Ola
Ringnér, Markus
Saal, Lao H.
Gruvberger-Saal, Sofia K.
Lövgren, Kristina
Grabau, Dorthe
Fernö, Mårten
Borg, Åke
Hegardt, Cecilia
SpringerLink (Online service)

DOI
10.1186/bcr2108
URN
urn:nbn:de:1111-201603241291
Rechteinformation
Der Zugriff auf das Objekt ist unbeschränkt möglich.
Letzte Aktualisierung
14.08.2025, 11:01 MESZ

Datenpartner

Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Original beim Datenpartner anzeigen

Beteiligte

  • Honeth, Gabriella
  • Bendahl, Pär-Ola
  • Ringnér, Markus
  • Saal, Lao H.
  • Gruvberger-Saal, Sofia K.
  • Lövgren, Kristina
  • Grabau, Dorthe
  • Fernö, Mårten
  • Borg, Åke
  • Hegardt, Cecilia
  • SpringerLink (Online service)

Ähnliche Objekte (12)

CD44/CD24 Expression in recurrent gastric cancer: a retrospective analysis

CD44/CD24 Expression in recurrent gastric cancer: a retrospective analysis

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

CCL3 predicts exceptional response to TGFβ inhibition in basal-like pancreatic cancer enriched in LIF-producing macrophages

CCL3 predicts exceptional response to TGFβ inhibition in basal-like pancreatic cancer enriched in LIF-producing macrophages

CD44/CD24 immunophenotypes on clinicopathologic features of salivary glands malignant neoplasms

CD44/CD24 immunophenotypes on clinicopathologic features of salivary glands malignant neoplasms

CD44 enhances invasion of basal-like breast cancer cells by upregulating serine protease and collagen-degrading enzymatic expression and activity

CD44 enhances invasion of basal-like breast cancer cells by upregulating serine protease and collagen-degrading enzymatic expression and activity

Immunodetection of cells with a CD44+/CD24- phenotype in canine mammary neoplasms

Immunodetection of cells with a CD44+/CD24- phenotype in canine mammary neoplasms

Differential expression of HIF-1α in CD44+CD24-/low breast ductal carcinomas

Differential expression of HIF-1α in CD44+CD24-/low breast ductal carcinomas

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Weight loss reduces basal-like breast cancer through kinome reprogramming

Weight loss reduces basal-like breast cancer through kinome reprogramming

SLUG/SNAI2 and Tumor Necrosis Factor Generate Breast Cells With CD44+/CD24- Phenotype

SLUG/SNAI2 and Tumor Necrosis Factor Generate Breast Cells With CD44+/CD24- Phenotype

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Verbundene Objekte