The glomerular parietal epithelial cell’s responses are influenced by SM22 alpha levels
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1471-2369
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
The glomerular parietal epithelial cell’s responses are influenced by SM22 alpha levels ; volume:15 ; number:1 ; day:6 ; month:11 ; year:2014 ; pages:1-11 ; date:12.2014
BMC nephrology ; 15, Heft 1 (6.11.2014), 1-11, 12.2014
- Creator
-
Naito, Shokichi
- Contributor
-
Pippin, Jeffrey W.
Shankland, Stuart J.
SpringerLink (Online service)
- DOI
-
10.1186/1471-2369-15-174
- URN
-
urn:nbn:de:1111-201608133718
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:45 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Naito, Shokichi
- Pippin, Jeffrey W.
- Shankland, Stuart J.
- SpringerLink (Online service)
Other Objects (12)
Investigations of activated parietal epithelial cells in glomerular disease
Primary cultures of glomerular parietal epithelial cells or podocytes with proven origin
Parietal cells—new perspectives in glomerular disease
Novel parietal epithelial cell subpopulations contribute to focal segmental glomerulosclerosis and glomerular tip lesions
The tetraspanin CD9 controls migration and proliferation of parietal epithelial cells and glomerular disease progression
The tetraspanin CD9 controls migration and proliferation of parietal epithelial cells and glomerular disease progression
Parietal epithelial cell dysfunction in crescentic glomerulonephritis
Perihilar FSGS lesions originate from flat parietal epithelial cells
Perihilar FSGS lesions originate from flat parietal epithelial cells
Parietal epithelial cells maintain the epithelial cell continuum forming Bowman's space in focal segmental glomerulosclerosis
Modulating parietal epithelial cell activation by inhibiting the JNK pathway and depleting macrophages
The recruitment mechanisms and potential therapeutic targets of podocytes from parietal epithelial cells
Investigations of activated parietal epithelial cells in glomerular disease
Primary cultures of glomerular parietal epithelial cells or podocytes with proven origin
Parietal cells—new perspectives in glomerular disease
Novel parietal epithelial cell subpopulations contribute to focal segmental glomerulosclerosis and glomerular tip lesions
The tetraspanin CD9 controls migration and proliferation of parietal epithelial cells and glomerular disease progression
The tetraspanin CD9 controls migration and proliferation of parietal epithelial cells and glomerular disease progression
Parietal epithelial cell dysfunction in crescentic glomerulonephritis
Perihilar FSGS lesions originate from flat parietal epithelial cells
Perihilar FSGS lesions originate from flat parietal epithelial cells
Parietal epithelial cells maintain the epithelial cell continuum forming Bowman's space in focal segmental glomerulosclerosis
Modulating parietal epithelial cell activation by inhibiting the JNK pathway and depleting macrophages
The recruitment mechanisms and potential therapeutic targets of podocytes from parietal epithelial cells
Investigations of activated parietal epithelial cells in glomerular disease
Primary cultures of glomerular parietal epithelial cells or podocytes with proven origin
Parietal cells—new perspectives in glomerular disease
Novel parietal epithelial cell subpopulations contribute to focal segmental glomerulosclerosis and glomerular tip lesions
The tetraspanin CD9 controls migration and proliferation of parietal epithelial cells and glomerular disease progression
The tetraspanin CD9 controls migration and proliferation of parietal epithelial cells and glomerular disease progression
Parietal epithelial cell dysfunction in crescentic glomerulonephritis
Perihilar FSGS lesions originate from flat parietal epithelial cells
Perihilar FSGS lesions originate from flat parietal epithelial cells
Parietal epithelial cells maintain the epithelial cell continuum forming Bowman's space in focal segmental glomerulosclerosis
Modulating parietal epithelial cell activation by inhibiting the JNK pathway and depleting macrophages