Author Correction: The epigenetic modifier Fam208a is required to maintain epiblast cell fitness

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2045-2322

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Author Correction: The epigenetic modifier Fam208a is required to maintain epiblast cell fitness ; volume:8 ; number:1 ; day:5 ; month:4 ; year:2018 ; pages:1 ; date:12.2018
Scientific reports ; 8, Heft 1 (5.4.2018), 1, 12.2018

Classification: Biowissenschaften, Biologie

Creator: Bhargava, Shohag

Contributor: Cox, Brian
Polydorou, Christiana
Gresakova, Veronika
Korinek, Vladimir
Strnad, Hynek
Sedlacek, Radislav
Epp, Trevor Allan
Chawengsaksophak, Kallayanee
SpringerLink (Online service)

DOI: 10.1038/s41598-018-22650-w

URN: urn:nbn:de:101:1-2018061016211576015826

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:34 AM CEST

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Associated

Bhargava, Shohag
Cox, Brian
Polydorou, Christiana
Gresakova, Veronika
Korinek, Vladimir
Strnad, Hynek
Sedlacek, Radislav
Epp, Trevor Allan
Chawengsaksophak, Kallayanee
SpringerLink (Online service)

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Stem cells tightly regulate dead cell clearance to maintain tissue fitness

The epigenetic modifier Fam208a is required to maintain epiblast cell fitness

Protein methylation is required to maintain optimal HIV-1 infectivity

MTGR1 is required to maintain small intestinal stem cell populations

Weak antagonistic fitness effects can maintain an inversion polymorphism

Combined epigenetic/genetic study identified an ALS age of onset modifier

Hochschulschrift

Nesprin-1 is required to maintain genomic stability and prevent tumorigenesis

Lineage-specific regulation of epigenetic modifier genes in human liver and brain

Oocyte TET3: an epigenetic modifier responsible for maternal inheritance of glucose intolerance

Cyproheptadine, an epigenetic modifier, exhibits anti-tumor activity by reversing the epigenetic silencing of IRF6 in urothelial carcinoma

EPB41L4A-AS1 is required to maintain basal autophagy to modulates Aβ clearance

Nrf2 is required to maintain the self-renewal of glioma stem cells

Stem cells tightly regulate dead cell clearance to maintain tissue fitness

The epigenetic modifier Fam208a is required to maintain epiblast cell fitness

Protein methylation is required to maintain optimal HIV-1 infectivity

MTGR1 is required to maintain small intestinal stem cell populations

Weak antagonistic fitness effects can maintain an inversion polymorphism

Combined epigenetic/genetic study identified an ALS age of onset modifier

Hochschulschrift

Nesprin-1 is required to maintain genomic stability and prevent tumorigenesis

Lineage-specific regulation of epigenetic modifier genes in human liver and brain

Oocyte TET3: an epigenetic modifier responsible for maternal inheritance of glucose intolerance

Cyproheptadine, an epigenetic modifier, exhibits anti-tumor activity by reversing the epigenetic silencing of IRF6 in urothelial carcinoma

EPB41L4A-AS1 is required to maintain basal autophagy to modulates Aβ clearance

Nrf2 is required to maintain the self-renewal of glioma stem cells

Stem cells tightly regulate dead cell clearance to maintain tissue fitness

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Author Correction: The epigenetic modifier Fam208a is required to maintain epiblast cell fitness

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Other Objects (12)

The epigenetic modifier Fam208a is required to maintain epiblast cell fitness

Protein methylation is required to maintain optimal HIV-1 infectivity

MTGR1 is required to maintain small intestinal stem cell populations

Weak antagonistic fitness effects can maintain an inversion polymorphism

Combined epigenetic/genetic study identified an ALS age of onset modifier

Nesprin-1 is required to maintain genomic stability and prevent tumorigenesis

Lineage-specific regulation of epigenetic modifier genes in human liver and brain

Oocyte TET3: an epigenetic modifier responsible for maternal inheritance of glucose intolerance

Cyproheptadine, an epigenetic modifier, exhibits anti-tumor activity by reversing the epigenetic silencing of IRF6 in urothelial carcinoma

EPB41L4A-AS1 is required to maintain basal autophagy to modulates Aβ clearance

Nrf2 is required to maintain the self-renewal of glioma stem cells

Stem cells tightly regulate dead cell clearance to maintain tissue fitness

The epigenetic modifier Fam208a is required to maintain epiblast cell fitness

Protein methylation is required to maintain optimal HIV-1 infectivity

MTGR1 is required to maintain small intestinal stem cell populations

Weak antagonistic fitness effects can maintain an inversion polymorphism

Combined epigenetic/genetic study identified an ALS age of onset modifier

Nesprin-1 is required to maintain genomic stability and prevent tumorigenesis

Lineage-specific regulation of epigenetic modifier genes in human liver and brain

Oocyte TET3: an epigenetic modifier responsible for maternal inheritance of glucose intolerance

Cyproheptadine, an epigenetic modifier, exhibits anti-tumor activity by reversing the epigenetic silencing of IRF6 in urothelial carcinoma

EPB41L4A-AS1 is required to maintain basal autophagy to modulates Aβ clearance

Nrf2 is required to maintain the self-renewal of glioma stem cells

Stem cells tightly regulate dead cell clearance to maintain tissue fitness

The epigenetic modifier Fam208a is required to maintain epiblast cell fitness

Protein methylation is required to maintain optimal HIV-1 infectivity

MTGR1 is required to maintain small intestinal stem cell populations

Weak antagonistic fitness effects can maintain an inversion polymorphism

Combined epigenetic/genetic study identified an ALS age of onset modifier

Nesprin-1 is required to maintain genomic stability and prevent tumorigenesis

Lineage-specific regulation of epigenetic modifier genes in human liver and brain

Oocyte TET3: an epigenetic modifier responsible for maternal inheritance of glucose intolerance

Cyproheptadine, an epigenetic modifier, exhibits anti-tumor activity by reversing the epigenetic silencing of IRF6 in urothelial carcinoma

EPB41L4A-AS1 is required to maintain basal autophagy to modulates Aβ clearance

Nrf2 is required to maintain the self-renewal of glioma stem cells

Stem cells tightly regulate dead cell clearance to maintain tissue fitness

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