- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1742-4690
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Protein methylation is required to maintain optimal HIV-1 infectivity ; volume:3 ; number:1 ; day:15 ; month:12 ; year:2006 ; pages:1-15 ; date:12.2006
Retrovirology ; 3, Heft 1 (15.12.2006), 1-15, 12.2006
- Creator
-
Willemsen, Nicole M.
- Contributor
-
Hitchen, Eleanor M.
Bodetti, Tracey J.
Apolloni, Ann
Warrilow, David
Piller, Sabine C.
Harrich, David
SpringerLink (Online service)
- DOI
-
10.1186/1742-4690-3-92
- URN
-
urn:nbn:de:1111-2016080654746
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:53 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Willemsen, Nicole M.
- Hitchen, Eleanor M.
- Bodetti, Tracey J.
- Apolloni, Ann
- Warrilow, David
- Piller, Sabine C.
- Harrich, David
- SpringerLink (Online service)
Other Objects (12)
HIV-1 Vif inhibits G to A hypermutations catalyzed by virus-encapsidated APOBEC3G to maintain HIV-1 infectivity
The developmental regulator PKL is required to maintain correct DNA methylation patterns at RNA-directed DNA methylation loci
A U5 repressor of reverse transcription is required for optimal HIV-1 infectivity and replication
Retraction Note to: A U5 repressor of reverse transcription is required for optimal HIV-1 infectivity and replication
The dimerization domain of HIV-1 viral infectivity factor Vif is required to block virion incorporation of APOBEC3G
MTGR1 is required to maintain small intestinal stem cell populations
Host factors modulating HIV-1 infectivity and restriction
The autophagy protein ATG9A promotes HIV-1 infectivity
Nesprin-1 is required to maintain genomic stability and prevent tumorigenesis
The epigenetic modifier Fam208a is required to maintain epiblast cell fitness
Nef antagonism of HIV-1 infectivity restriction and sensing
EPB41L4A-AS1 is required to maintain basal autophagy to modulates Aβ clearance
HIV-1 Vif inhibits G to A hypermutations catalyzed by virus-encapsidated APOBEC3G to maintain HIV-1 infectivity
The developmental regulator PKL is required to maintain correct DNA methylation patterns at RNA-directed DNA methylation loci
A U5 repressor of reverse transcription is required for optimal HIV-1 infectivity and replication
Retraction Note to: A U5 repressor of reverse transcription is required for optimal HIV-1 infectivity and replication
The dimerization domain of HIV-1 viral infectivity factor Vif is required to block virion incorporation of APOBEC3G
MTGR1 is required to maintain small intestinal stem cell populations
Host factors modulating HIV-1 infectivity and restriction
The autophagy protein ATG9A promotes HIV-1 infectivity
Nesprin-1 is required to maintain genomic stability and prevent tumorigenesis
The epigenetic modifier Fam208a is required to maintain epiblast cell fitness
Nef antagonism of HIV-1 infectivity restriction and sensing
EPB41L4A-AS1 is required to maintain basal autophagy to modulates Aβ clearance
HIV-1 Vif inhibits G to A hypermutations catalyzed by virus-encapsidated APOBEC3G to maintain HIV-1 infectivity
The developmental regulator PKL is required to maintain correct DNA methylation patterns at RNA-directed DNA methylation loci
A U5 repressor of reverse transcription is required for optimal HIV-1 infectivity and replication
Retraction Note to: A U5 repressor of reverse transcription is required for optimal HIV-1 infectivity and replication
The dimerization domain of HIV-1 viral infectivity factor Vif is required to block virion incorporation of APOBEC3G
MTGR1 is required to maintain small intestinal stem cell populations
Host factors modulating HIV-1 infectivity and restriction
The autophagy protein ATG9A promotes HIV-1 infectivity
Nesprin-1 is required to maintain genomic stability and prevent tumorigenesis
The epigenetic modifier Fam208a is required to maintain epiblast cell fitness
Nef antagonism of HIV-1 infectivity restriction and sensing