Transcriptomic changes underlying EGFR inhibitor resistance in human and mouse models of basal-like breast cancer

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2045-2322

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Transcriptomic changes underlying EGFR inhibitor resistance in human and mouse models of basal-like breast cancer ; volume:12 ; number:1 ; day:8 ; month:12 ; year:2022 ; pages:1-13 ; date:12.2022
Scientific reports ; 12, Heft 1 (8.12.2022), 1-13, 12.2022

Creator: Rashid, Narmeen S.
Boyd, David C.
Olex, Amy L.
Grible, Jacqueline M.
Duong, Alex K.
Alzubi, Mohammad A.
Altman, Julia E.
Leftwich, Tess J.
Valentine, Aaron D.
Hairr, Nicole S.
Zboril, Emily K.
Smith, Timothy M.
Pfefferle, Adam D.
Dozmorov, Mikhail G.
Harrell, J. Chuck

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41598-022-25541-3

URN: urn:nbn:de:101:1-2023021809104130460146

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:53 AM CEST

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Rashid, Narmeen S.
Boyd, David C.
Olex, Amy L.
Grible, Jacqueline M.
Duong, Alex K.
Alzubi, Mohammad A.
Altman, Julia E.
Leftwich, Tess J.
Valentine, Aaron D.
Hairr, Nicole S.
Zboril, Emily K.
Smith, Timothy M.
Pfefferle, Adam D.
Dozmorov, Mikhail G.
Harrell, J. Chuck
SpringerLink (Online service)

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Transcriptomic changes underlying EGFR inhibitor resistance in human and mouse models of basal-like breast cancer

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Other Objects (12)

ERβ1 represses basal-like breast cancer epithelial to mesenchymal transition by destabilizing EGFR

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Ductal keratin 15+ luminal progenitors in normal breast exhibit a basal-like breast cancer transcriptomic signature

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Weight loss reduces basal-like breast cancer through kinome reprogramming

Integrated molecular pathway analysis informs a synergistic combination therapy targeting PTEN/PI3K and EGFR pathways for basal-like breast cancer

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

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Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

ERβ1 represses basal-like breast cancer epithelial to mesenchymal transition by destabilizing EGFR

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Ductal keratin 15+ luminal progenitors in normal breast exhibit a basal-like breast cancer transcriptomic signature

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

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Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

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ME1 promotes basal-like breast cancer progression and associates with poor prognosis

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Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

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