METTL3 promotes glycolysis and cholangiocarcinoma progression by mediating the m6A modification of AKR1B10
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1475-2867
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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METTL3 promotes glycolysis and cholangiocarcinoma progression by mediating the m6A modification of AKR1B10 ; volume:22 ; number:1 ; day:7 ; month:12 ; year:2022 ; pages:1-18 ; date:12.2022
Cancer cell international ; 22, Heft 1 (7.12.2022), 1-18, 12.2022
- Urheber
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Cai, Jingli
Cui, Zheng
Zhou, Jingyi
Zhang, Bosen
Lu, Ruiqi
Ding, Youcheng
Hu, Hai
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1186/s12935-022-02809-2
- URN
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urn:nbn:de:101:1-2023021721053198832986
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:51 MESZ
Datenpartner
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Beteiligte
- Cai, Jingli
- Cui, Zheng
- Zhou, Jingyi
- Zhang, Bosen
- Lu, Ruiqi
- Ding, Youcheng
- Hu, Hai
- SpringerLink (Online service)
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Mettl3-mediated mRNA m6A methylation promotes dendritic cell activation
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METTL3-mediated m6A mRNA modification of FBXW7 suppresses lung adenocarcinoma
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METTL3, m6A modification, and EGR1: interplay affecting myocardial I/R injury outcomes