METTL3 promotes glycolysis and cholangiocarcinoma progression by mediating the m6A modification of AKR1B10
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1475-2867
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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METTL3 promotes glycolysis and cholangiocarcinoma progression by mediating the m6A modification of AKR1B10 ; volume:22 ; number:1 ; day:7 ; month:12 ; year:2022 ; pages:1-18 ; date:12.2022
Cancer cell international ; 22, Heft 1 (7.12.2022), 1-18, 12.2022
- Creator
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Cai, Jingli
Cui, Zheng
Zhou, Jingyi
Zhang, Bosen
Lu, Ruiqi
Ding, Youcheng
Hu, Hai
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12935-022-02809-2
- URN
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urn:nbn:de:101:1-2023021721053198832986
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:51 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Cai, Jingli
- Cui, Zheng
- Zhou, Jingyi
- Zhang, Bosen
- Lu, Ruiqi
- Ding, Youcheng
- Hu, Hai
- SpringerLink (Online service)
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Novel insights into the m6A-RNA methyltransferase METTL3 in cancer
METTL3-mediated m6A mRNA modification of FBXW7 suppresses lung adenocarcinoma
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Mettl3-dependent m6A modification attenuates the brain stress response in Drosophila
Dynamic m6A mRNA methylation reveals the role of METTL3-m6A-CDCP1 signaling axis in chemical carcinogenesis
METTL3 promotes colorectal carcinoma progression by regulating the m6A–CRB3–Hippo axis
METTL3, m6A modification, and EGR1: interplay affecting myocardial I/R injury outcomes
The N6-methyladenosine METTL3 regulates tumorigenesis and glycolysis by mediating m6A methylation of the tumor suppressor LATS1 in breast cancer
The methyltransferase METTL3 promotes tumorigenesis via mediating HHLA2 mRNA m6A modification in human renal cell carcinoma
m6A-dependent glycolysis enhances colorectal cancer progression
Mettl3-mediated mRNA m6A methylation promotes dendritic cell activation
Novel insights into the m6A-RNA methyltransferase METTL3 in cancer
METTL3-mediated m6A mRNA modification of FBXW7 suppresses lung adenocarcinoma
METTL3-mediated m6A modification increases Hspa1a stability to inhibit osteoblast aging
Mettl3-dependent m6A modification attenuates the brain stress response in Drosophila
Mettl3-dependent m6A modification attenuates the brain stress response in Drosophila
Dynamic m6A mRNA methylation reveals the role of METTL3-m6A-CDCP1 signaling axis in chemical carcinogenesis
METTL3 promotes colorectal carcinoma progression by regulating the m6A–CRB3–Hippo axis
METTL3, m6A modification, and EGR1: interplay affecting myocardial I/R injury outcomes
The N6-methyladenosine METTL3 regulates tumorigenesis and glycolysis by mediating m6A methylation of the tumor suppressor LATS1 in breast cancer
The methyltransferase METTL3 promotes tumorigenesis via mediating HHLA2 mRNA m6A modification in human renal cell carcinoma
m6A-dependent glycolysis enhances colorectal cancer progression
Mettl3-mediated mRNA m6A methylation promotes dendritic cell activation
Novel insights into the m6A-RNA methyltransferase METTL3 in cancer
METTL3-mediated m6A mRNA modification of FBXW7 suppresses lung adenocarcinoma
METTL3-mediated m6A modification increases Hspa1a stability to inhibit osteoblast aging
Mettl3-dependent m6A modification attenuates the brain stress response in Drosophila
Mettl3-dependent m6A modification attenuates the brain stress response in Drosophila
Dynamic m6A mRNA methylation reveals the role of METTL3-m6A-CDCP1 signaling axis in chemical carcinogenesis
METTL3 promotes colorectal carcinoma progression by regulating the m6A–CRB3–Hippo axis