- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD ; volume:10 ; number:1 ; year:2018 ; pages:4-6 ; extent:3
EMBO molecular medicine / European Molecular Biology Organization ; 10, Heft 1 (2018), 4-6 (gesamt 3)
- Creator
- DOI
-
10.15252/emmm.201708572
- URN
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urn:nbn:de:101:1-2022083007251599105347
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:35 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Schludi, Martin H.
- Edbauer, Dieter
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Sense and antisense RNA are not toxic in Drosophila models of C9orf72-associated ALS/FTD
A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma
Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit
Retention of hexanucleotide repeat-containing intron in C9orf72 mRNA: implications for the pathogenesis of ALS/FTD
A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD
Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD
Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell
Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD
Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies
RNA dependent suppression of C9orf72 ALS/FTD associated neurodegeneration by Matrin-3
Phenylalanine-tRNA aminoacylation is compromised by ALS/FTD-associated C9orf72 C4G2 repeat RNA
The ALS/FTD-related C9orf72 hexanucleotide repeat expansion forms RNA condensates through multimolecular G-quadruplexes
Sense and antisense RNA are not toxic in Drosophila models of C9orf72-associated ALS/FTD
A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma
Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit
Retention of hexanucleotide repeat-containing intron in C9orf72 mRNA: implications for the pathogenesis of ALS/FTD
A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD
Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD
Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell
Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD
Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies
RNA dependent suppression of C9orf72 ALS/FTD associated neurodegeneration by Matrin-3
Phenylalanine-tRNA aminoacylation is compromised by ALS/FTD-associated C9orf72 C4G2 repeat RNA
The ALS/FTD-related C9orf72 hexanucleotide repeat expansion forms RNA condensates through multimolecular G-quadruplexes
Sense and antisense RNA are not toxic in Drosophila models of C9orf72-associated ALS/FTD
A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma
Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit
Retention of hexanucleotide repeat-containing intron in C9orf72 mRNA: implications for the pathogenesis of ALS/FTD
A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD
Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD
Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell
Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD