- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1750-1326
- Extent
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Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD ; volume:12 ; number:1 ; day:12 ; month:6 ; year:2017 ; pages:1-11 ; date:12.2017
Molecular neurodegeneration ; 12, Heft 1 (12.6.2017), 1-11, 12.2017
- Keyword
-
Histologie
Cytologie
- Creator
-
Esanov, Rustam
- Contributor
-
Cabrera, Gabriela Toro
Andrade, Nadja S.
Gendron, Tania F.
Brown, Robert H.
Benatar, Michael
Wahlestedt, Claes
Mueller, Christian
Zeier, Zane
SpringerLink (Online service)
- DOI
-
10.1186/s13024-017-0185-9
- URN
-
urn:nbn:de:1111-201709063344
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
23.03.20258025, 9:53 AM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Esanov, Rustam
- Cabrera, Gabriela Toro
- Andrade, Nadja S.
- Gendron, Tania F.
- Brown, Robert H.
- Benatar, Michael
- Wahlestedt, Claes
- Mueller, Christian
- Zeier, Zane
- SpringerLink (Online service)
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Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD
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Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies
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The ALS/FTD-related C9orf72 hexanucleotide repeat expansion forms RNA condensates through multimolecular G-quadruplexes
CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts
An interaction between synapsin and C9orf72 regulates excitatory synapses and is impaired in ALS/FTD
A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma
Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit
Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD
Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD
Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell
RNA dependent suppression of C9orf72 ALS/FTD associated neurodegeneration by Matrin-3
Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD
Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies
Phenylalanine-tRNA aminoacylation is compromised by ALS/FTD-associated C9orf72 C4G2 repeat RNA
The ALS/FTD-related C9orf72 hexanucleotide repeat expansion forms RNA condensates through multimolecular G-quadruplexes
CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts
An interaction between synapsin and C9orf72 regulates excitatory synapses and is impaired in ALS/FTD
A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma
Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit
Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD
Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD
Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell
RNA dependent suppression of C9orf72 ALS/FTD associated neurodegeneration by Matrin-3
Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD
Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies
Phenylalanine-tRNA aminoacylation is compromised by ALS/FTD-associated C9orf72 C4G2 repeat RNA
The ALS/FTD-related C9orf72 hexanucleotide repeat expansion forms RNA condensates through multimolecular G-quadruplexes
CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts