Different roles of integrin-β1 and integrin-αv for type IV secretion of CagA versus cell elongation phenotype and cell lifting by Helicobacter pylori
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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In: PLoS Pathogens 16.7 (2020).
- Event
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Veröffentlichung
- (where)
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Erlangen
- (who)
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Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU)
- (when)
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2020
- Creator
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Tegtmeyer, Nicole
Backert, Steffen
- DOI
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10.1371/journal.ppat.1008135
- URN
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urn:nbn:de:bvb:29-opus4-142564
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 3:19 PM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Tegtmeyer, Nicole
- Backert, Steffen
- Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU)
Time of origin
- 2020
Other Objects (12)
Type IV Secretion and Signal Transduction of Helicobacter pylori CagA through Interactions with Host Cell Receptors
Molecular mechanisms of gastric epithelial cell adhesion and injection of CagA by Helicobacter pylori
Helicobacter pylori CagL Y58/E59 Mutation Turns-Off Type IV Secretion-Dependent Delivery of CagA into Host Cells
Helicobacter pylori CagA Induces Cortactin Y-470 Phosphorylation-Dependent Gastric Epithelial Cell Scattering via Abl, Vav2 and Rac1 Activation
The Helicobacter pylori type IV secretion system upregulates epithelial cortactin expression by a CagA‐ and JNK‐dependent pathway
Cortactin Promotes Effective AGS Cell Scattering by Helicobacter pylori CagA, but Not Cellular Vacuolization and Apoptosis Induced by the Vacuolating Cytotoxin VacA
Overexpression of serine protease HtrA enhances disruption of adherens junctions, paracellular transmigration and type IV secretion of CagA by Helicobacter pylori
Overexpression of serine protease HtrA enhances disruption of adherens junctions, paracellular transmigration and type IV secretion of CagA by Helicobacter pylori
A Specific A/T Polymorphism in Western Tyrosine Phosphorylation B-Motifs Regulates Helicobacter pylori CagA Epithelial Cell Interactions
Untersuchungen zur Struktur und Replikation des Mitochondriengenoms von Chenopodium album (L.)
Integrin-independent Helicobacter pylori CagA translocation via type IV secretion
Helicobacter pylori controls NLRP3 expression by regulating hsa-miR-223-3p and IL-10 in cultured and primary human immune cells
Type IV Secretion and Signal Transduction of Helicobacter pylori CagA through Interactions with Host Cell Receptors
Molecular mechanisms of gastric epithelial cell adhesion and injection of CagA by Helicobacter pylori
Helicobacter pylori CagL Y58/E59 Mutation Turns-Off Type IV Secretion-Dependent Delivery of CagA into Host Cells
Helicobacter pylori CagA Induces Cortactin Y-470 Phosphorylation-Dependent Gastric Epithelial Cell Scattering via Abl, Vav2 and Rac1 Activation
The Helicobacter pylori type IV secretion system upregulates epithelial cortactin expression by a CagA‐ and JNK‐dependent pathway
Cortactin Promotes Effective AGS Cell Scattering by Helicobacter pylori CagA, but Not Cellular Vacuolization and Apoptosis Induced by the Vacuolating Cytotoxin VacA
Overexpression of serine protease HtrA enhances disruption of adherens junctions, paracellular transmigration and type IV secretion of CagA by Helicobacter pylori
Overexpression of serine protease HtrA enhances disruption of adherens junctions, paracellular transmigration and type IV secretion of CagA by Helicobacter pylori
A Specific A/T Polymorphism in Western Tyrosine Phosphorylation B-Motifs Regulates Helicobacter pylori CagA Epithelial Cell Interactions
Untersuchungen zur Struktur und Replikation des Mitochondriengenoms von Chenopodium album (L.)
Integrin-independent Helicobacter pylori CagA translocation via type IV secretion
Helicobacter pylori controls NLRP3 expression by regulating hsa-miR-223-3p and IL-10 in cultured and primary human immune cells
Type IV Secretion and Signal Transduction of Helicobacter pylori CagA through Interactions with Host Cell Receptors
Molecular mechanisms of gastric epithelial cell adhesion and injection of CagA by Helicobacter pylori
Helicobacter pylori CagL Y58/E59 Mutation Turns-Off Type IV Secretion-Dependent Delivery of CagA into Host Cells
Helicobacter pylori CagA Induces Cortactin Y-470 Phosphorylation-Dependent Gastric Epithelial Cell Scattering via Abl, Vav2 and Rac1 Activation
The Helicobacter pylori type IV secretion system upregulates epithelial cortactin expression by a CagA‐ and JNK‐dependent pathway
Cortactin Promotes Effective AGS Cell Scattering by Helicobacter pylori CagA, but Not Cellular Vacuolization and Apoptosis Induced by the Vacuolating Cytotoxin VacA
Overexpression of serine protease HtrA enhances disruption of adherens junctions, paracellular transmigration and type IV secretion of CagA by Helicobacter pylori
Overexpression of serine protease HtrA enhances disruption of adherens junctions, paracellular transmigration and type IV secretion of CagA by Helicobacter pylori
A Specific A/T Polymorphism in Western Tyrosine Phosphorylation B-Motifs Regulates Helicobacter pylori CagA Epithelial Cell Interactions
Untersuchungen zur Struktur und Replikation des Mitochondriengenoms von Chenopodium album (L.)
Integrin-independent Helicobacter pylori CagA translocation via type IV secretion