Chromatin accessibility and transcription dynamics during in vitro astrocyte differentiation of Huntington’s Disease Monkey pluripotent stem cells
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1756-8935
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Chromatin accessibility and transcription dynamics during in vitro astrocyte differentiation of Huntington’s Disease Monkey pluripotent stem cells ; volume:12 ; number:1 ; day:13 ; month:11 ; year:2019 ; pages:1-25 ; date:12.2019
Epigenetics & chromatin ; 12, Heft 1 (13.11.2019), 1-25, 12.2019
- Creator
-
Goodnight, Alexandra V.
Kremsky, Isaac
Khampang, Sujittra
Jung, Yoon Hee
Billingsley, James M.
Bosinger, Steven E.
Corces, Victor G.
Chan, Anthony W. S.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s13072-019-0313-6
- URN
-
urn:nbn:de:101:1-2020031119133718313512
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:47 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Goodnight, Alexandra V.
- Kremsky, Isaac
- Khampang, Sujittra
- Jung, Yoon Hee
- Billingsley, James M.
- Bosinger, Steven E.
- Corces, Victor G.
- Chan, Anthony W. S.
- SpringerLink (Online service)
Other Objects (12)
Multi-omic analysis of Huntington’s disease reveals a compensatory astrocyte state
Single-nucleus RNA-seq identifies Huntington disease astrocyte states
Huntington
Manifestation of Huntington’s disease pathology in human induced pluripotent stem cell-derived neurons
Monkey hybrid stem cells develop cellular features of Huntington's disease
Mechanism suppressing proteostasis collapse in pluripotent stem cells and its demise in Huntington’s disease
Astrocytes generated from patient induced pluripotent stem cells recapitulate features of Huntington’s disease patient cells
The ubiquitin ligase UBR5 suppresses proteostasis collapse in pluripotent stem cells from Huntington’s disease patients
A two years longitudinal study of a transgenic Huntington disease monkey
Huntington's chorea
Chorea Huntington
Huntington-Erkrankung
Multi-omic analysis of Huntington’s disease reveals a compensatory astrocyte state
Single-nucleus RNA-seq identifies Huntington disease astrocyte states
Huntington
Manifestation of Huntington’s disease pathology in human induced pluripotent stem cell-derived neurons
Monkey hybrid stem cells develop cellular features of Huntington's disease
Mechanism suppressing proteostasis collapse in pluripotent stem cells and its demise in Huntington’s disease
Astrocytes generated from patient induced pluripotent stem cells recapitulate features of Huntington’s disease patient cells
The ubiquitin ligase UBR5 suppresses proteostasis collapse in pluripotent stem cells from Huntington’s disease patients
A two years longitudinal study of a transgenic Huntington disease monkey
Huntington's chorea
Chorea Huntington
Huntington-Erkrankung
Multi-omic analysis of Huntington’s disease reveals a compensatory astrocyte state
Single-nucleus RNA-seq identifies Huntington disease astrocyte states
Huntington
Manifestation of Huntington’s disease pathology in human induced pluripotent stem cell-derived neurons
Monkey hybrid stem cells develop cellular features of Huntington's disease
Mechanism suppressing proteostasis collapse in pluripotent stem cells and its demise in Huntington’s disease
Astrocytes generated from patient induced pluripotent stem cells recapitulate features of Huntington’s disease patient cells
The ubiquitin ligase UBR5 suppresses proteostasis collapse in pluripotent stem cells from Huntington’s disease patients
A two years longitudinal study of a transgenic Huntington disease monkey
Huntington's chorea
Chorea Huntington