Veratridine Can Bind to a Site at the Mouth of the Channel Pore at Human Cardiac Sodium Channel NaV1.5
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Erschienen in
-
In: International Journal of Molecular Sciences. 23
- Ereignis
-
Veröffentlichung
- (wo)
-
Leipzig
- (wer)
-
Universitätsbibliothek Leipzig
- (wann)
-
2024
- Urheber
-
Gulsevin, Alican
Glazer, Andrew M.
Shields, Tiffany
Kroncke, Brett M.
Roden, Dan M.
Meiler, Jens
- URN
-
urn:nbn:de:bsz:15-qucosa2-891656
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.03.2025, 13:51 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Gulsevin, Alican
- Glazer, Andrew M.
- Shields, Tiffany
- Kroncke, Brett M.
- Roden, Dan M.
- Meiler, Jens
- Universitätsbibliothek Leipzig
Entstanden
- 2024
Ähnliche Objekte (12)
Sodium channel slow inactivation interferes with open channel block
Pharmacological modification of sodium channel Nav1.9 activity
Earthquake at the Devil`s Channel
Uncoupling sodium channel dimers restores the phenotype of a pain‐linked Nav1.7 channel mutation
Molecular modeling of human voltage-gated sodium channel complexes
The "Brisk", Telegraph-Ship at her Moorings in the Channel
Magnetohydrodynamic duct and channel flows at finite magnetic Reynolds numbers
Channel Entente
Channel Entente
Channel Entente
Channel Entente
Channel Entente
Sodium channel slow inactivation interferes with open channel block
Pharmacological modification of sodium channel Nav1.9 activity
Earthquake at the Devil`s Channel
Uncoupling sodium channel dimers restores the phenotype of a pain‐linked Nav1.7 channel mutation
Molecular modeling of human voltage-gated sodium channel complexes
The "Brisk", Telegraph-Ship at her Moorings in the Channel
Magnetohydrodynamic duct and channel flows at finite magnetic Reynolds numbers
Channel Entente
Channel Entente
Channel Entente
Channel Entente
Channel Entente
Sodium channel slow inactivation interferes with open channel block
Pharmacological modification of sodium channel Nav1.9 activity
Earthquake at the Devil`s Channel
Uncoupling sodium channel dimers restores the phenotype of a pain‐linked Nav1.7 channel mutation
Molecular modeling of human voltage-gated sodium channel complexes
The "Brisk", Telegraph-Ship at her Moorings in the Channel
Magnetohydrodynamic duct and channel flows at finite magnetic Reynolds numbers
Channel Entente
Channel Entente
Channel Entente
Channel Entente