- Alternative title
-
Der Drosophila-Nephrozyt - Ein Modell zur Podozytenfunktion und -erkrankung
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Regensburg, Universität Regensburg, Dissertation, 2018
- Keyword
-
Drosophila
- Event
-
Veröffentlichung
- (where)
-
Regensburg
- (who)
-
Universitätsbibliothek Regensburg
- (when)
-
2019
- Creator
-
Hochapfel, Florian
- Contributor
-
Krahn, Michael
- URN
-
urn:nbn:de:bvb:355-epub-406764
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:22 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Hochapfel, Florian
- Krahn, Michael
- Universitätsbibliothek Regensburg
Time of origin
- 2019
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Role of microRNAs in podocyte structure and function
Glomerular podocyte dysfunction in inherited renal tubular disease
Histone Deacetylases Take Center Stage on Regulation of Podocyte Function
Nuclear YAP localization as a key regulator of podocyte function
mTOR-dependent autophagy regulates slit diaphragm density in podocyte-like drosophila nephrocytes
TRPC5-mediated podocyte calcium toxicity drives progressive glomerular disease
TRPC5-mediated podocyte calcium toxicity drives progressive glomerular disease
Podocyte GSK3 is an evolutionarily conserved critical regulator of kidney function
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Using the drosophila nephrocyte to model podocyte function and disease
Podocyte OTUD5 alleviates diabetic kidney disease through deubiquitinating TAK1 and reducing podocyte inflammation and injury
The transcription factor Dach1 is essential for podocyte function
Role of microRNAs in podocyte structure and function
Glomerular podocyte dysfunction in inherited renal tubular disease
Histone Deacetylases Take Center Stage on Regulation of Podocyte Function
Nuclear YAP localization as a key regulator of podocyte function
mTOR-dependent autophagy regulates slit diaphragm density in podocyte-like drosophila nephrocytes
TRPC5-mediated podocyte calcium toxicity drives progressive glomerular disease
TRPC5-mediated podocyte calcium toxicity drives progressive glomerular disease
Podocyte GSK3 is an evolutionarily conserved critical regulator of kidney function