CD8+ lymphocyte infiltration is an independent favorable prognostic indicator in basal-like breast cancer
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1465-542X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
CD8+ lymphocyte infiltration is an independent favorable prognostic indicator in basal-like breast cancer ; volume:14 ; number:2 ; day:15 ; month:3 ; year:2012 ; pages:1-14 ; date:4.2012
Breast cancer research ; 14, Heft 2 (15.3.2012), 1-14, 4.2012
- Creator
-
Liu, Shuzhen
- Contributor
-
Lachapelle, Jonathan
Leung, Samuel
Gao, Dongxia
Foulkes, William D.
Nielsen, Torsten O.
SpringerLink (Online service)
- DOI
-
10.1186/bcr3148
- URN
-
urn:nbn:de:1111-2016032414952
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:45 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Liu, Shuzhen
- Lachapelle, Jonathan
- Leung, Samuel
- Gao, Dongxia
- Foulkes, William D.
- Nielsen, Torsten O.
- SpringerLink (Online service)
Other Objects (12)
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Weight loss reduces basal-like breast cancer through kinome reprogramming
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Gene expression signatures of morphologically normal breast tissue identify basal-like tumors
Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer
ME1 promotes basal-like breast cancer progression and associates with poor prognosis
MFAP5 promotes basal-like breast cancer progression by activating the EMT program
Increased gene expression variability in BRCA1-associated and basal-like breast tumours
Metabolic biomarkers for response to PI3K inhibition in basal-like breast cancer
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Weight loss reduces basal-like breast cancer through kinome reprogramming
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Gene expression signatures of morphologically normal breast tissue identify basal-like tumors
Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer
ME1 promotes basal-like breast cancer progression and associates with poor prognosis
MFAP5 promotes basal-like breast cancer progression by activating the EMT program
Increased gene expression variability in BRCA1-associated and basal-like breast tumours
Metabolic biomarkers for response to PI3K inhibition in basal-like breast cancer
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Weight loss reduces basal-like breast cancer through kinome reprogramming
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Gene expression signatures of morphologically normal breast tissue identify basal-like tumors
Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer
ME1 promotes basal-like breast cancer progression and associates with poor prognosis
MFAP5 promotes basal-like breast cancer progression by activating the EMT program
Increased gene expression variability in BRCA1-associated and basal-like breast tumours