Dual role of HDAC10 in lysosomal exocytosis and DNA repair promotes neuroblastoma chemoresistance

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2045-2322

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Dual role of HDAC10 in lysosomal exocytosis and DNA repair promotes neuroblastoma chemoresistance ; volume:8 ; number:1 ; day:3 ; month:7 ; year:2018 ; pages:1-17 ; date:12.2018
Scientific reports ; 8, Heft 1 (3.7.2018), 1-17, 12.2018

Creator: Ridinger, Johannes

Contributor: Koeneke, Emily
Kolbinger, Fiona R.
Koerholz, Katharina
Mahboobi, Siavosh
Hellweg, Lars
Gunkel, Nikolas
Miller, Aubry K.
Peterziel, Heike
Schmezer, Peter
Hamacher-Brady, Anne
Witt, Olaf
Oehme, Ina
SpringerLink (Online service)

DOI: 10.1038/s41598-018-28265-5

URN: urn:nbn:de:101:1-2018090223141130097617

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:24 AM CEST

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Associated

Ridinger, Johannes
Koeneke, Emily
Kolbinger, Fiona R.
Koerholz, Katharina
Mahboobi, Siavosh
Hellweg, Lars
Gunkel, Nikolas
Miller, Aubry K.
Peterziel, Heike
Schmezer, Peter
Hamacher-Brady, Anne
Witt, Olaf
Oehme, Ina
SpringerLink (Online service)

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Dual role of HDAC10 in lysosomal exocytosis and DNA repair promotes neuroblastoma chemoresistance

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Other Objects (12)

HDAC10 in neuroblastoma chemoresistance

Autophagy is associated with chemoresistance in neuroblastoma

Lysosomal exocytosis releases pathogenic α-synuclein species from neurons in synucleinopathy models

Gene expression signature of acquired chemoresistance in neuroblastoma cells

TNF-α promotes α-synuclein propagation through stimulation of senescence-associated lysosomal exocytosis

Correction: TNF-α promotes α-synuclein propagation through stimulation of senescence-associated lysosomal exocytosis

Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance

Mechanisms of regulated exocytosis in plasma membrane repair and endothelial cell secretion

Early Endosomes Act as Local Exocytosis Hubs to Repair Endothelial Membrane Damage

Integrin α3β1 promotes vessel formation of glioblastoma-associated endothelial cells through calcium-mediated macropinocytosis and lysosomal exocytosis

Inducin Triggers LC3‐Lipidation and ESCRT‐Mediated Lysosomal Membrane Repair

Insulin granule biogenesis and exocytosis

HDAC10 in neuroblastoma chemoresistance

Autophagy is associated with chemoresistance in neuroblastoma

Lysosomal exocytosis releases pathogenic α-synuclein species from neurons in synucleinopathy models

Gene expression signature of acquired chemoresistance in neuroblastoma cells

TNF-α promotes α-synuclein propagation through stimulation of senescence-associated lysosomal exocytosis

Correction: TNF-α promotes α-synuclein propagation through stimulation of senescence-associated lysosomal exocytosis

Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance

Mechanisms of regulated exocytosis in plasma membrane repair and endothelial cell secretion

Early Endosomes Act as Local Exocytosis Hubs to Repair Endothelial Membrane Damage

Integrin α3β1 promotes vessel formation of glioblastoma-associated endothelial cells through calcium-mediated macropinocytosis and lysosomal exocytosis

Inducin Triggers LC3‐Lipidation and ESCRT‐Mediated Lysosomal Membrane Repair

Insulin granule biogenesis and exocytosis

HDAC10 in neuroblastoma chemoresistance

Autophagy is associated with chemoresistance in neuroblastoma

Lysosomal exocytosis releases pathogenic α-synuclein species from neurons in synucleinopathy models

Gene expression signature of acquired chemoresistance in neuroblastoma cells

TNF-α promotes α-synuclein propagation through stimulation of senescence-associated lysosomal exocytosis

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