Autophagy is associated with chemoresistance in neuroblastoma

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1471-2407

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Autophagy is associated with chemoresistance in neuroblastoma ; volume:16 ; number:1 ; day:15 ; month:11 ; year:2016 ; pages:1-14 ; date:12.2016
BMC cancer ; 16, Heft 1 (15.11.2016), 1-14, 12.2016

Keyword: Krebs
Onkologie

Creator: Belounis, Assila

Contributor: Nyalendo, Carine
Le Gall, Roxane
Imbriglio, Tina V.
Mahma, Mohamed
Teira, Pierre
Beaunoyer, Mona
Cournoyer, Sonia
Haddad, Elie
Vassal, Gilles
Sartelet, Hervé
SpringerLink (Online service)

DOI: 10.1186/s12885-016-2906-9

URN: urn:nbn:de:1111-201703204496

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:57 AM CEST

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Associated

Belounis, Assila
Nyalendo, Carine
Le Gall, Roxane
Imbriglio, Tina V.
Mahma, Mohamed
Teira, Pierre
Beaunoyer, Mona
Cournoyer, Sonia
Haddad, Elie
Vassal, Gilles
Sartelet, Hervé
SpringerLink (Online service)

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HDAC10 in neuroblastoma chemoresistance

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Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (−)-Gossypol (AT-101)

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Gene expression signature of acquired chemoresistance in neuroblastoma cells

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Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (−)-Gossypol (AT-101)

Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (-)-Gossypol (AT-101)

Coordinated autophagy modulation overcomes glioblastoma chemoresistance through disruption of mitochondrial bioenergetics

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Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance

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Autophagy is associated with chemoresistance in neuroblastoma

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Other Objects (12)

HDAC10 in neuroblastoma chemoresistance

Gene expression signature of acquired chemoresistance in neuroblastoma cells

Dual role of HDAC10 in lysosomal exocytosis and DNA repair promotes neuroblastoma chemoresistance

Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (−)-Gossypol (AT-101)

Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (-)-Gossypol (AT-101)

Coordinated autophagy modulation overcomes glioblastoma chemoresistance through disruption of mitochondrial bioenergetics

Long noncoding RNA MALAT1 regulates autophagy associated chemoresistance via miR-23b-3p sequestration in gastric cancer

Chemoresistance acquisition induces a global shift of expression of aniogenesis-associated genes and increased pro-angogenic activity in neuroblastoma cells

Chemoresistance acquisition induces a global shift of expression of aniogenesis-associated genes and increased pro-angogenic activity in neuroblastoma cells

Transcriptional regulation of autophagy : identification of autophagy regulators as potential effectors of targeted therapy in neuroblastoma

Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance

RAC3 influences the chemoresistance of colon cancer cells through autophagy and apoptosis inhibition

HDAC10 in neuroblastoma chemoresistance

Gene expression signature of acquired chemoresistance in neuroblastoma cells

Dual role of HDAC10 in lysosomal exocytosis and DNA repair promotes neuroblastoma chemoresistance

Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (−)-Gossypol (AT-101)

Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (-)-Gossypol (AT-101)

Coordinated autophagy modulation overcomes glioblastoma chemoresistance through disruption of mitochondrial bioenergetics

Long noncoding RNA MALAT1 regulates autophagy associated chemoresistance via miR-23b-3p sequestration in gastric cancer

Chemoresistance acquisition induces a global shift of expression of aniogenesis-associated genes and increased pro-angogenic activity in neuroblastoma cells

Chemoresistance acquisition induces a global shift of expression of aniogenesis-associated genes and increased pro-angogenic activity in neuroblastoma cells

Transcriptional regulation of autophagy : identification of autophagy regulators as potential effectors of targeted therapy in neuroblastoma

Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance

RAC3 influences the chemoresistance of colon cancer cells through autophagy and apoptosis inhibition

HDAC10 in neuroblastoma chemoresistance

Gene expression signature of acquired chemoresistance in neuroblastoma cells

Dual role of HDAC10 in lysosomal exocytosis and DNA repair promotes neuroblastoma chemoresistance

Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (−)-Gossypol (AT-101)

Chemoresistance is associated with increased cytoprotective autophagy and diminished apoptosis in bladder cancer cells treated with the BH3 mimetic (-)-Gossypol (AT-101)

Coordinated autophagy modulation overcomes glioblastoma chemoresistance through disruption of mitochondrial bioenergetics

Long noncoding RNA MALAT1 regulates autophagy associated chemoresistance via miR-23b-3p sequestration in gastric cancer

Chemoresistance acquisition induces a global shift of expression of aniogenesis-associated genes and increased pro-angogenic activity in neuroblastoma cells

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Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance

RAC3 influences the chemoresistance of colon cancer cells through autophagy and apoptosis inhibition

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