Hochschulschrift
The influence of specific mitochondrial polymorphisms on the α-synuclein-induced pathology in a mouse model of Parkinson's disease
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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Magdeburg, Universität, Diss., 2015
- Keyword
-
Hirnkrankheit
Neuropsychiatrie
Nervenkrankheit
- Event
-
Veröffentlichung
- (where)
-
Magdeburg
- (who)
-
Universitätsbibliothek
- (when)
-
2015
- Creator
- URN
-
urn:nbn:de:gbv:ma9:1-6736
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:54 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Schwitlick, Christina
- Universitätsbibliothek
Time of origin
- 2015
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Alpha-synuclein overexpression in the olfactory bulb initiates prodromal symptoms and pathology of Parkinson’s disease
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Linking acetylated α-Tubulin redistribution to α-Synuclein pathology in brain of Parkinson’s disease patients
α-Synuclein pathology disrupts mitochondrial function in dopaminergic and cholinergic neurons at-risk in Parkinson’s disease
Initiation and progression of α-synuclein pathology in Parkinson’s disease
Mitochondrial dynamics in alpha-synuclein model of Parkinson`s disease
Association of Glial Activation and α-Synuclein Pathology in Parkinson’s Disease
The relationship between α-synuclein [alpha-synuclein] and the proteasome and its role in Parkinson's pathology
Mitochondrial DNA damage triggers spread of Parkinson’s disease-like pathology
α-Synuclein pathology in Parkinson disease activates homeostatic NRF2 anti-oxidant response
Discrepancy between distribution of alpha-synuclein oligomers and Lewy-related pathology in Parkinson’s disease
Blocking meningeal lymphatic drainage aggravates Parkinson’s disease-like pathology in mice overexpressing mutated α-synuclein
Alpha-synuclein overexpression in the olfactory bulb initiates prodromal symptoms and pathology of Parkinson’s disease
Ischemic stroke causes Parkinson’s disease-like pathology and symptoms in transgenic mice overexpressing alpha-synuclein
Linking acetylated α-Tubulin redistribution to α-Synuclein pathology in brain of Parkinson’s disease patients
α-Synuclein pathology disrupts mitochondrial function in dopaminergic and cholinergic neurons at-risk in Parkinson’s disease
Initiation and progression of α-synuclein pathology in Parkinson’s disease
Mitochondrial dynamics in alpha-synuclein model of Parkinson`s disease
Association of Glial Activation and α-Synuclein Pathology in Parkinson’s Disease
The relationship between α-synuclein [alpha-synuclein] and the proteasome and its role in Parkinson's pathology
Mitochondrial DNA damage triggers spread of Parkinson’s disease-like pathology
α-Synuclein pathology in Parkinson disease activates homeostatic NRF2 anti-oxidant response
Discrepancy between distribution of alpha-synuclein oligomers and Lewy-related pathology in Parkinson’s disease
Blocking meningeal lymphatic drainage aggravates Parkinson’s disease-like pathology in mice overexpressing mutated α-synuclein
Alpha-synuclein overexpression in the olfactory bulb initiates prodromal symptoms and pathology of Parkinson’s disease
Ischemic stroke causes Parkinson’s disease-like pathology and symptoms in transgenic mice overexpressing alpha-synuclein