Molecular analysis reveals heterogeneity of mouse mammary tumors conditionally mutant for Brca1

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1476-4598

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Molecular analysis reveals heterogeneity of mouse mammary tumors conditionally mutant for Brca1 ; volume:7 ; number:1 ; day:07 ; month:4 ; year:2008 ; pages:1-12 ; date:12.2008
Molecular cancer ; 7, Heft 1 (07.4.2008), 1-12, 12.2008

Creator: Wright, Mollie H.
Robles, Ana I.
Herschkowitz, Jason I.
Hollingshead, Melinda G.
Anver, Miriam R.
Perou, Charles M.
Varticovski, Lyuba

Contributor: SpringerLink (Online service)

DOI: 10.1186/1476-4598-7-29

URN: urn:nbn:de:101:1-2021082218550624973291

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:01 AM CEST

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Associated

Wright, Mollie H.
Robles, Ana I.
Herschkowitz, Jason I.
Hollingshead, Melinda G.
Anver, Miriam R.
Perou, Charles M.
Varticovski, Lyuba
SpringerLink (Online service)

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BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis

Loss of BRCA1 leads to an increase in epidermal growth factor receptor expression in mammary epithelial cells, and epidermal growth factor receptor inhibition prevents estrogen receptor-negative cancers in BRCA1-mutant mice

Cyclin B1 stability is increased by interaction with BRCA1, and its overexpression suppresses the progression of BRCA1-associated mammary tumors

Multipotent luminal mammary cancer stem cells model tumor heterogeneity

Patient-derived conditionally reprogrammed cells maintain intra-tumor genetic heterogeneity

BRCA1 mutation influences progesterone response in human benign mammary organoids

Conditionally specified distributions

Therapeutic targeting of BRCA1 and TP53 mutant breast cancer through mutant p53 reactivation

Heterogeneity of mammary lesions represent molecular differences

PDGFRβ is an essential therapeutic target for BRCA1-deficient mammary tumors

BRCA1 Deficiency: BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis (Adv. Sci. 6/2020)

Conditionally approximately convex functions

BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis

Loss of BRCA1 leads to an increase in epidermal growth factor receptor expression in mammary epithelial cells, and epidermal growth factor receptor inhibition prevents estrogen receptor-negative cancers in BRCA1-mutant mice

Cyclin B1 stability is increased by interaction with BRCA1, and its overexpression suppresses the progression of BRCA1-associated mammary tumors

Multipotent luminal mammary cancer stem cells model tumor heterogeneity

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Molecular analysis reveals heterogeneity of mouse mammary tumors conditionally mutant for Brca1

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Other Objects (12)

Patient-derived conditionally reprogrammed cells maintain intra-tumor genetic heterogeneity

BRCA1 mutation influences progesterone response in human benign mammary organoids

Conditionally specified distributions

Therapeutic targeting of BRCA1 and TP53 mutant breast cancer through mutant p53 reactivation

Heterogeneity of mammary lesions represent molecular differences

PDGFRβ is an essential therapeutic target for BRCA1-deficient mammary tumors

BRCA1 Deficiency: BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis (Adv. Sci. 6/2020)

Conditionally approximately convex functions

BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis

Loss of BRCA1 leads to an increase in epidermal growth factor receptor expression in mammary epithelial cells, and epidermal growth factor receptor inhibition prevents estrogen receptor-negative cancers in BRCA1-mutant mice

Cyclin B1 stability is increased by interaction with BRCA1, and its overexpression suppresses the progression of BRCA1-associated mammary tumors

Multipotent luminal mammary cancer stem cells model tumor heterogeneity

Patient-derived conditionally reprogrammed cells maintain intra-tumor genetic heterogeneity

BRCA1 mutation influences progesterone response in human benign mammary organoids

Conditionally specified distributions

Therapeutic targeting of BRCA1 and TP53 mutant breast cancer through mutant p53 reactivation

Heterogeneity of mammary lesions represent molecular differences

PDGFRβ is an essential therapeutic target for BRCA1-deficient mammary tumors

BRCA1 Deficiency: BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis (Adv. Sci. 6/2020)

Conditionally approximately convex functions

BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis

Loss of BRCA1 leads to an increase in epidermal growth factor receptor expression in mammary epithelial cells, and epidermal growth factor receptor inhibition prevents estrogen receptor-negative cancers in BRCA1-mutant mice

Cyclin B1 stability is increased by interaction with BRCA1, and its overexpression suppresses the progression of BRCA1-associated mammary tumors

Multipotent luminal mammary cancer stem cells model tumor heterogeneity

Patient-derived conditionally reprogrammed cells maintain intra-tumor genetic heterogeneity

BRCA1 mutation influences progesterone response in human benign mammary organoids

Conditionally specified distributions

Therapeutic targeting of BRCA1 and TP53 mutant breast cancer through mutant p53 reactivation

Heterogeneity of mammary lesions represent molecular differences

PDGFRβ is an essential therapeutic target for BRCA1-deficient mammary tumors

BRCA1 Deficiency: BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis (Adv. Sci. 6/2020)

Conditionally approximately convex functions

BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis

Loss of BRCA1 leads to an increase in epidermal growth factor receptor expression in mammary epithelial cells, and epidermal growth factor receptor inhibition prevents estrogen receptor-negative cancers in BRCA1-mutant mice

Cyclin B1 stability is increased by interaction with BRCA1, and its overexpression suppresses the progression of BRCA1-associated mammary tumors

Multipotent luminal mammary cancer stem cells model tumor heterogeneity

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