Cyclin B1 stability is increased by interaction with BRCA1, and its overexpression suppresses the progression of BRCA1-associated mammary tumors

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2092-6413

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Cyclin B1 stability is increased by interaction with BRCA1, and its overexpression suppresses the progression of BRCA1-associated mammary tumors ; volume:50 ; number:10 ; day:16 ; month:10 ; year:2018 ; pages:1-16 ; date:10.2018
Experimental & molecular medicine ; 50, Heft 10 (16.10.2018), 1-16, 10.2018

Creator: Choi, Eun Kyung

Contributor: Lim, Jeong-A
Kim, Jong Kwang
Jang, Moon Sun
Kim, Sun Eui
Baek, Hye Jung
Park, Eun Jung
Kim, Tae Hyun
Deng, Chu-Xia
Wang, Rui-Hong
Kim, Sang Soo
SpringerLink (Online service)

DOI: 10.1038/s12276-018-0169-z

URN: urn:nbn:de:101:1-2018112120313279235199

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:46 AM CEST

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Associated

Choi, Eun Kyung
Lim, Jeong-A
Kim, Jong Kwang
Jang, Moon Sun
Kim, Sun Eui
Baek, Hye Jung
Park, Eun Jung
Kim, Tae Hyun
Deng, Chu-Xia
Wang, Rui-Hong
Kim, Sang Soo
SpringerLink (Online service)

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Cyclin B1 stability is increased by interaction with BRCA1, and its overexpression suppresses the progression of BRCA1-associated mammary tumors

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Other Objects (12)

BRCA1 Deficiency: BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis (Adv. Sci. 6/2020)

BRCA1 mutation influences progesterone response in human benign mammary organoids

Activation of FGFR2 Signaling Suppresses BRCA1 and Drives Triple‐Negative Mammary Tumorigenesis That is Sensitive to Immunotherapy

Activation of FGFR2 Signaling Suppresses BRCA1 and Drives Triple‐Negative Mammary Tumorigenesis That is Sensitive to Immunotherapy

PDGFRβ is an essential therapeutic target for BRCA1-deficient mammary tumors

Molecular analysis reveals heterogeneity of mouse mammary tumors conditionally mutant for Brca1

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Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers

Proliferationsaktivität von BRCA1-assoziierten Mammakarzinomen

BRCA1 Deficiency: BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis (Adv. Sci. 6/2020)

BRCA1 mutation influences progesterone response in human benign mammary organoids

Activation of FGFR2 Signaling Suppresses BRCA1 and Drives Triple‐Negative Mammary Tumorigenesis That is Sensitive to Immunotherapy

Activation of FGFR2 Signaling Suppresses BRCA1 and Drives Triple‐Negative Mammary Tumorigenesis That is Sensitive to Immunotherapy

PDGFRβ is an essential therapeutic target for BRCA1-deficient mammary tumors

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BRCA1 testing in Lithuania

Gene-Specific Genetic Complementation between Brca1 and Cobra1 During Mouse Mammary Gland Development

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Embryonic mammary signature subsets are activated in Brca1 -/- and basal-like breast cancers

Proliferationsaktivität von BRCA1-assoziierten Mammakarzinomen

BRCA1 Deficiency: BRCA1 Deficiency Impairs Mitophagy and Promotes Inflammasome Activation and Mammary Tumor Metastasis (Adv. Sci. 6/2020)

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Activation of FGFR2 Signaling Suppresses BRCA1 and Drives Triple‐Negative Mammary Tumorigenesis That is Sensitive to Immunotherapy

Activation of FGFR2 Signaling Suppresses BRCA1 and Drives Triple‐Negative Mammary Tumorigenesis That is Sensitive to Immunotherapy

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Proliferationsaktivität von BRCA1-assoziierten Mammakarzinomen

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