Angiotensin-(1-9) prevents cardiomyocyte hypertrophy via miR-129-3p/PKIA/PKA signaling pathway
- Alternative title
-
Angiotensin-(1-9) verhindert Kardiomyozyten-Hypertrophie über miR-129-3p/PKIA/PKA-Signalweg
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Regensburg, Universität Regensburg, Dissertation, 2021
- Keyword
-
Hypertrophy
Sotomayor
Regensburg
- Event
-
Veröffentlichung
- (where)
-
Regensburg
- (who)
-
Universitätsbibliothek Regensburg
- (when)
-
2022
- Creator
- Contributor
-
Meister, Gunter
- URN
-
urn:nbn:de:bvb:355-epub-510048
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:42 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sotomayor Flores, Cristian
- Meister, Gunter
- Universitätsbibliothek Regensburg
Time of origin
- 2022
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Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
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Using iPSC-CMs to develop a new, 2D in vitro culture-based model for cardiomyocyte hypertrophy
PKA
PKA
Amlexanox and forskolin prevents isoproterenol-induced cardiomyopathy by subduing cardiomyocyte hypertrophy and maladaptive inflammatory responses
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
Three-dimensional human pluripotent stem cell-derived cardiomyocyte models for investigating cardiac hypertrophy
Inhibition of Nuclear Import of Calcineurin Prevents the Development of Myocardial Hypertrophy
Cardiomyocyte hyperplasia and immaturity but not hypertrophy are characteristic features of patients with RASopathies
Altered cellular protein quality control system modulates cardiomyocyte function in volume overload-induced hypertrophy
Long non-coding RNA Malat1 deficiency prevents neonatal heart regeneration by inducing cardiomyocyte binucleation
Using iPSC-CMs to develop a new, 2D in vitro culture-based model for cardiomyocyte hypertrophy
PKA
PKA
Amlexanox and forskolin prevents isoproterenol-induced cardiomyopathy by subduing cardiomyocyte hypertrophy and maladaptive inflammatory responses
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
Three-dimensional human pluripotent stem cell-derived cardiomyocyte models for investigating cardiac hypertrophy
Inhibition of Nuclear Import of Calcineurin Prevents the Development of Myocardial Hypertrophy
Cardiomyocyte hyperplasia and immaturity but not hypertrophy are characteristic features of patients with RASopathies
Altered cellular protein quality control system modulates cardiomyocyte function in volume overload-induced hypertrophy
Long non-coding RNA Malat1 deficiency prevents neonatal heart regeneration by inducing cardiomyocyte binucleation
Using iPSC-CMs to develop a new, 2D in vitro culture-based model for cardiomyocyte hypertrophy
PKA