Angiotensin-(1-9) prevents cardiomyocyte hypertrophy via miR-129-3p/PKIA/PKA signaling pathway
- Alternative title
-
Angiotensin-(1-9) verhindert Kardiomyozyten-Hypertrophie über miR-129-3p/PKIA/PKA-Signalweg
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Regensburg, Universität Regensburg, Dissertation, 2021
- Keyword
-
Hypertrophy
Sotomayor
Regensburg
- Event
-
Veröffentlichung
- (where)
-
Regensburg
- (who)
-
Universitätsbibliothek Regensburg
- (when)
-
2022
- Creator
- Contributor
-
Meister, Gunter
- URN
-
urn:nbn:de:bvb:355-epub-510048
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:30 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sotomayor Flores, Cristian
- Meister, Gunter
- Universitätsbibliothek Regensburg
Time of origin
- 2022
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Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation
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Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival
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LOC102549726/miR-760-3p network is involved in the progression of ISO-induced pathological cardiomyocyte hypertrophy via endoplasmic reticulum stress
ADAP1 limits neonatal cardiomyocyte hypertrophy by reducing integrin cell surface expression
Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy
MRI based non-invasive detection of cardiomyocyte hypertrophy and cell-volume changes
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Proteomic analysis of neonatal mouse hearts shows PKA functions as a cardiomyocyte replication regulator
Amlexanox and forskolin prevents isoproterenol-induced cardiomyopathy by subduing cardiomyocyte hypertrophy and maladaptive inflammatory responses
Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Circular RNA circZFPM2 regulates cardiomyocyte hypertrophy and survival
The functional role of m6A demethylase ALKBH5 in cardiomyocyte hypertrophy
LOC102549726/miR-760-3p network is involved in the progression of ISO-induced pathological cardiomyocyte hypertrophy via endoplasmic reticulum stress
ADAP1 limits neonatal cardiomyocyte hypertrophy by reducing integrin cell surface expression
Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy
MRI based non-invasive detection of cardiomyocyte hypertrophy and cell-volume changes
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy