Examining the association between serum galactose-deficient IgA1 and primary IgA nephropathy: a systematic review and meta-analysis
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
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Examining the association between serum galactose-deficient IgA1 and primary IgA nephropathy: a systematic review and meta-analysis ; day:1 ; month:3 ; year:2024 ; pages:1-14
Journal of nephrology ; (1.3.2024), 1-14
- Creator
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Vaz de Castro, Pedro Alves Soares
Amaral, Arthur Aguiar
Almeida, Mariana Godinho
Selvaskandan, Haresh
Barratt, Jonathan
Simões e Silva, Ana Cristina
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s40620-023-01874-8
- URN
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urn:nbn:de:101:1-2405052123280.404615517309
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:54 AM CEST
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Associated
- Vaz de Castro, Pedro Alves Soares
- Amaral, Arthur Aguiar
- Almeida, Mariana Godinho
- Selvaskandan, Haresh
- Barratt, Jonathan
- Simões e Silva, Ana Cristina
- SpringerLink (Online service)
Other Objects (12)
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
Leukemia Inhibitory Factor Signaling Enhances Production of Galactose-Deficient IgA1 in IgA Nephropathy
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
Mesangial cells from patients with IgA nephropathy have increased susceptibility to galactose-deficient IgA1
Renal pathological analysis using galactose-deficient IgA1-specific monoclonal antibody is a strong tool for differentiation of primary IgA nephropathy from secondary IgA nephropathy
Serum levels of galactose-deficient IgA are elevated in patients with IgA nephropathy but do not correlate to disease activity or progression
Sialylation of IgG inhibits the formation of galactose-deficient IgA1-containing immune complexes and protects mesangial cells from injury in IgA nephropathy
Comprehensive analysis of aberrantly expressed profiles of mRNA and its relationship with serum galactose-deficient IgA1 level in IgA nephropathy
IgA nephropathy today : 10 tables
Current treatment of IgA nephropathy
Complement activation in IgA nephropathy
IgA Nephropathy: A Chinese Perspective
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
Leukemia Inhibitory Factor Signaling Enhances Production of Galactose-Deficient IgA1 in IgA Nephropathy
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
Mesangial cells from patients with IgA nephropathy have increased susceptibility to galactose-deficient IgA1
Renal pathological analysis using galactose-deficient IgA1-specific monoclonal antibody is a strong tool for differentiation of primary IgA nephropathy from secondary IgA nephropathy
Serum levels of galactose-deficient IgA are elevated in patients with IgA nephropathy but do not correlate to disease activity or progression
Sialylation of IgG inhibits the formation of galactose-deficient IgA1-containing immune complexes and protects mesangial cells from injury in IgA nephropathy
Comprehensive analysis of aberrantly expressed profiles of mRNA and its relationship with serum galactose-deficient IgA1 level in IgA nephropathy
IgA nephropathy today : 10 tables
Current treatment of IgA nephropathy
Complement activation in IgA nephropathy
IgA Nephropathy: A Chinese Perspective
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
Leukemia Inhibitory Factor Signaling Enhances Production of Galactose-Deficient IgA1 in IgA Nephropathy
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
Mesangial cells from patients with IgA nephropathy have increased susceptibility to galactose-deficient IgA1
Renal pathological analysis using galactose-deficient IgA1-specific monoclonal antibody is a strong tool for differentiation of primary IgA nephropathy from secondary IgA nephropathy
Serum levels of galactose-deficient IgA are elevated in patients with IgA nephropathy but do not correlate to disease activity or progression
Sialylation of IgG inhibits the formation of galactose-deficient IgA1-containing immune complexes and protects mesangial cells from injury in IgA nephropathy
Comprehensive analysis of aberrantly expressed profiles of mRNA and its relationship with serum galactose-deficient IgA1 level in IgA nephropathy
IgA nephropathy today : 10 tables
Current treatment of IgA nephropathy
Complement activation in IgA nephropathy