Sialylation of IgG inhibits the formation of galactose-deficient IgA1-containing immune complexes and protects mesangial cells from injury in IgA nephropathy
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2369
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Sialylation of IgG inhibits the formation of galactose-deficient IgA1-containing immune complexes and protects mesangial cells from injury in IgA nephropathy ; volume:23 ; number:1 ; day:11 ; month:1 ; year:2022 ; pages:1-9 ; date:12.2022
BMC nephrology ; 23, Heft 1 (11.1.2022), 1-9, 12.2022
- Creator
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Liu, Youxia
Li, Hongfen
Yu, Huyan
Wang, Fanghao
Jia, Junya
Yan, Tiekun
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12882-021-02657-8
- URN
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urn:nbn:de:101:1-2022042518560339591188
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:34 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Liu, Youxia
- Li, Hongfen
- Yu, Huyan
- Wang, Fanghao
- Jia, Junya
- Yan, Tiekun
- SpringerLink (Online service)
Other Objects (12)
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Renal pathological analysis using galactose-deficient IgA1-specific monoclonal antibody is a strong tool for differentiation of primary IgA nephropathy from secondary IgA nephropathy
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Immunglobulinsubklassen bei monoklonalen IgG- und IgA-Gammopathien
Mesangial cells from patients with IgA nephropathy have increased susceptibility to galactose-deficient IgA1
IgG subclass deposition in diabetic nephropathy
Role of Human Mesangial-Tubular Crosstalk in Secretory IgA-Induced IgA Nephropathy
Pathogenic role of glycan-specific IgG antibodies in IgA nephropathy
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
Leukemia Inhibitory Factor Signaling Enhances Production of Galactose-Deficient IgA1 in IgA Nephropathy
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
IgE, IgG und IgG [IgG-IgE] als klinisch-therapeutische Parameter bei verschiedenen Hyposensibilisierungsmethoden der Pollinose
Normwertevaluierung fetaler Immunglobuline IgA, IgM, IgE, IgG und IgG-Subklassen IgG 1 - 4 aus intrauterin gewonnenem Serum
Renal pathological analysis using galactose-deficient IgA1-specific monoclonal antibody is a strong tool for differentiation of primary IgA nephropathy from secondary IgA nephropathy
Serum IgA, IgM, and IgG responses in COVID-19
Immunglobulinsubklassen bei monoklonalen IgG- und IgA-Gammopathien
Mesangial cells from patients with IgA nephropathy have increased susceptibility to galactose-deficient IgA1
IgG subclass deposition in diabetic nephropathy
Role of Human Mesangial-Tubular Crosstalk in Secretory IgA-Induced IgA Nephropathy
Pathogenic role of glycan-specific IgG antibodies in IgA nephropathy
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
Leukemia Inhibitory Factor Signaling Enhances Production of Galactose-Deficient IgA1 in IgA Nephropathy
Clinical Significance of Galactose-Deficient IgA1 by KM55 in Patients with IgA Nephropathy
IgE, IgG und IgG [IgG-IgE] als klinisch-therapeutische Parameter bei verschiedenen Hyposensibilisierungsmethoden der Pollinose
Normwertevaluierung fetaler Immunglobuline IgA, IgM, IgE, IgG und IgG-Subklassen IgG 1 - 4 aus intrauterin gewonnenem Serum
Renal pathological analysis using galactose-deficient IgA1-specific monoclonal antibody is a strong tool for differentiation of primary IgA nephropathy from secondary IgA nephropathy
Serum IgA, IgM, and IgG responses in COVID-19